Author

J.-F. Pare

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 1 Immunology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Assay:bulk RNA-seq· 1 Research Focus:cell signaling & trafficking· 1 Research Focus:drug response / drug discovery· 1 Organism:human· 1 Organism:mouse· 1 Assay:proteomics· 1 Organism:Xenopus· 1

Frequent Co-Authors

A. Ghaffari· 1 shared A. L. Dinis· 1 shared A. W. B. Craig· 1 shared C. H. Graham· 1 shared C. L. Margerum· 1 shared D. E. Ingber· 1 shared D. M. Camacho· 1 shared D. R. Siemens· 1 shared

Activity by Year

2021-2023 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 24 mutual co-author links

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1 shared paperCancer Biology

A. Ghaffari

Mutual co-authors in cluster

Author profile →

2 papers

Recent·Most cited

Autophagy mediates cancer cell resistance to doxorubicin induced by the Programmed Death 1/Programmed Death Ligand 1 immune checkpoint axis

L. Minassian, D. Sanwalka, J.-F. Pare, S. Macdonald-Goodfellow, R. L. Sutherland, A. Ghaffari, C. L. Margerum, M. Koti, A. W. B. Craig, T. Cotechini, D. R. Siemens, E. Chan, C. H. Graham

This study shows that PD-1/PD-L1 signaling induces autophagy in cancer cells, contributing to drug resistance, and suggests combining PD-1/PD-L1 blockers with autophagy inhibitors could improve chemotherapy effectiveness.

bioRxiv · Jan 17, 2023 · PreprintCancer Biology

Enhancers of host immune tolerance to bacterial infection discovered using linked computational and experimental approaches

M. M. Sperry, R. Novak, V. Keshari, A. L. Dinis, M. J. Cartwright, D. M. Camacho, J.-F. Pare, M. Super, M. Levin, D. E. Ingber

The paper identifies host tolerance mechanisms to bacterial infection using a Xenopus model and multi-omics analysis, finding that metal chelators and HIF-1 agonists enhance survival.

bioRxiv · Sep 30, 2021Immunology