Author

K. Eschbach

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 1 Microbiology· 1

Signature Assays, Organisms & Methods

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Assay:bulk RNA-seq· 1 Research Focus:cell type annotation· 1 Research Focus:differential expression· 1 Approach:experimental assay· 1 Research Focus:host-pathogen & immune response· 1 Organism:human· 1 Organism:mouse· 1 Assay:proteomics· 1

Frequent Co-Authors

C. Beisel· 2 shared A. Banaei-Esfahani· 1 shared A. Sethi· 1 shared A. Trauner· 1 shared B. C. Collins· 1 shared B. Hamelin· 1 shared H. Kohler· 1 shared J. Feldmann· 1 shared

Activity by Year

2018-2022 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 25 mutual co-author links

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2 shared papersCancer Biology

C. Beisel

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Author profile →

2 papers

Recent·Most cited

Single-cell analysis reveals inter- and intratumour heterogeneity in metastatic breast cancer

B. Hamelin, M. M. S. Obradovic, A. Sethi, M. Koch, S. Muenst, C. Beisel, K. Eschbach, H. Kohler, S. Soysal, M. Vetter, W. P. Weber, M. Stadler, M. Bentires-Alj

This study uses single-cell RNA-Seq to identify molecular states and partial EMT phenotypes in breast cancer tumours and metastases, revealing common transcription factors across patients.

bioRxiv · Oct 16, 2022Cancer Biology

Resource misallocation as a mediator of fitness costs in antibiotic resistance.

A. Trauner, A. Banaei-Esfahani, S. M. Gygli, P. Warmer, J. Feldmann, S. Shafieechashmi, K. Eschbach, M. Zampieri, S. Borrell, B. C. Collins, C. Beisel, R. Aebersold, S. Gagneux

The paper investigates the fitness cost of rifampicin resistance in Mycobacterium tuberculosis using transcriptomic and proteomic profiling, finding that aberrant gene expression causes the cost and that this burden can be suppressed.

bioRxiv · Nov 27, 2018 · PreprintMicrobiology