Author

K. Rivera

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

Click to filter author papers

Cancer Biology· 2 Ecology· 1

Signature Assays, Organisms & Methods

Click to filter · or open tag

Approach:experimental assay· 2 Organism:human· 2 Approach:statistical model· 2 Assay:bulk RNA-seq· 1 Research Focus:cell signaling & trafficking· 1 Research Focus:cell type annotation· 1 Research Focus:ecology & biodiversity· 1 Research Focus:gene regulatory network inference· 1

Frequent Co-Authors

D. J. Pappin· 2 shared A. Murphy· 1 shared A. T. Yu· 1 shared B. Gerber· 1 shared B. Liu· 1 shared C. Berasain· 1 shared C. J. Logothetis· 1 shared D. L. Spector· 1 shared

Activity by Year

2021-2021 · peak 3/yr

Co-Author Collaboration Network

10 top collaborators · 19 mutual co-author links

Click any collaborator node to inspect or filter shared papers

Focal author (center)Selected co-authorORCID co-authorUnlinked name

Curved chords = mutual co-authorship

2 shared papersCancer Biology

D. J. Pappin

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

Prostate tumor-induced stromal reprogramming generates Tenascin C that promotes prostate cancer metastasis through YAP/TAZ inhibition

S.-H. Lin, Y.-C. Lee, S.-C. Lin, G. Yu, M. Zhu, J. H. Song, K. Rivera, D. J. Pappin, C. J. Logothetis, T. Panaretakis, G. Wang, L.-y. Yu-Lee

This study shows that prostate tumor-induced stromal reprogramming produces Tenascin C, which promotes metastasis by inhibiting YAP/TAZ through 5{beta}1 integrin.

bioRxiv · Nov 8, 2021Cancer Biology

Rethinking habitat occupancy modeling and the role of diel activity in an anthropogenic world

K. Rivera, M. Fidino, Z. Farris, S. Magle, A. Murphy, B. Gerber

The paper develops a new occupancy model to analyze diel activity patterns of species in response to human disturbance and finds that both fosa and coyote adjust their habitat use across diel states and years.

bioRxiv · Jun 30, 2021Ecology

PHAROH lncRNA regulates c-Myc translation in hepatocellular carcinoma via sequestering TIAR

A. T. Yu, C. Berasain, S. Bhatia, K. Rivera, B. Liu, F. Rigo, D. J. Pappin, D. L. Spector

The paper identifies PHAROH, an lncRNA overexpressed in hepatocellular carcinoma, which regulates c-Myc translation by sequestering TIAR, impacting cell proliferation and migration.

bioRxiv · Mar 23, 2021Cancer Biology