L. M. Ferreri

This study shows that influenza A virus escape from stem-binding antibodies incurs fitness costs, but epistatic interactions can mitigate these costs in some contexts.

This study shows that human-origin influenza A viruses can transmit in pigs and select for adaptive mutations in the HA gene that improve replication and fitness in swine hosts.

This study evaluates the consistency of ferret transmission experiments across laboratories for two H1N1 influenza viruses, finding concordant results and suggesting a minimum of three to five labs for reliable risk assessment.

This study examines reassortment dynamics of influenza A virus in guinea pigs, ferrets, and swine, finding reassortment is common but more frequent in ferrets and guinea pigs than swine, with spatial compartmentalization observed.

This study shows that the timing of SARS-CoV-2 exposure, particularly during peak viral load, is the strongest determinant of transmission success in golden Syrian hamsters.

The paper shows that influenza A virus requires collective interactions for replication in mammalian cells but not in avian cells, with the PA gene segment playing a key role.