K. Ganti

This study identifies viral expansion after transfer as a key factor in influenza A virus transmission bottlenecks, showing that diversity declines rapidly post-infection.

This study investigates how influenza A virus coinfection dynamics are influenced by virus-virus interactions within and between cells, finding both beneficial intracellular interactions and antagonistic host-level competition.

This study examines reassortment dynamics of influenza A virus in guinea pigs, ferrets, and swine, finding reassortment is common but more frequent in ferrets and guinea pigs than swine, with spatial compartmentalization observed.

This study shows that the timing of SARS-CoV-2 exposure, particularly during peak viral load, is the strongest determinant of transmission success in golden Syrian hamsters.

This study identifies Rab11a as a critical host factor for influenza A virus genome assembly by facilitating vRNP congregation and specific interactions.

This study compares avian influenza A virus reassortment in mallards and guinea pigs, finding higher reassortant frequencies and diversity in mallards.

This paper identifies Rab11a's role in facilitating cell-cell spread of influenza A virus genomes via tunneling nanotubes, enabling genome reassortment and bypassing extracellular barriers.

The paper shows that influenza A virus requires collective interactions for replication in mammalian cells but not in avian cells, with the PA gene segment playing a key role.

This study identifies dysregulation of M segment gene expression, particularly M2 overexpression, as a key factor in avian influenza A virus restriction in mammalian hosts.