Author

L. Morla

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Pathology· 1 Physiology· 1

Signature Assays, Organisms & Methods

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Research Focus:cell type annotation· 2 Approach:experimental assay· 2 Organism:human· 2 Assay:proteomics· 2 Research Focus:differential expression· 1 Research Focus:host-pathogen & immune response· 1 Research Focus:metabolism & bioenergetics· 1 Organism:mouse· 1

Frequent Co-Authors

A. Doucet· 2 shared C. Walter· 2 shared G. Crambert· 2 shared A. Blanchard· 1 shared A. Sassi· 1 shared B. Matot· 1 shared B. Vogt· 1 shared C. Rafael· 1 shared

Activity by Year

2021-2022 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 33 mutual co-author links

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2 shared papersPhysiology

A. Doucet

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Author profile →

2 papers

Recent·Most cited

GDF15 mediates renal cell plasticity in response to potassium depletion

S. Lasaad, C. Walter, C. Rafael, L. Morla, A. Doucet, N. Picard, A. Blanchard, Y. Fromes, B. Matot, G. Crambert, L. Cheval

This study shows that GDF15 mediates renal cell plasticity in response to potassium depletion by regulating intercalated cell numbers and K+ retention.

bioRxiv · Dec 27, 2022 · PreprintPhysiology

A new variant of ASIC2 mediates sodium retention in nephrotic syndrome

M. Fila, A. Sassi, G. Brideau, L. Morla, P. Houillier, C. Walter, M. Gennaoui, L. Collignon, M. Keck, G. Planelles, N. Bakouh, M. Peuchmaur, G. Deschenes, I. Anegon, S. Remy, , A. Doucet

The paper identifies a truncated variant of ASIC2b responsible for sodium retention in idiopathic nephrotic syndrome, independent of ENaC and aldosterone.

bioRxiv · Feb 9, 2021Pathology