Author

P. Houillier

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Pathology· 1 Pharmacology & Toxicology· 1

Signature Assays, Organisms & Methods

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Research Focus:cell type annotation· 2 Approach:experimental assay· 2 Organism:rat· 2 Research Focus:cell signaling & trafficking· 1 Research Focus:differential expression· 1 Organism:human· 1 Assay:imaging· 1 Research Focus:protein folding & dynamics· 1

Frequent Co-Authors

G. Crambert· 2 shared A. Doucet· 1 shared A. Sassi· 1 shared B. Megarbane· 1 shared B. Vogt· 1 shared C. Klein· 1 shared C. Walter· 1 shared G. Brideau· 1 shared

Activity by Year

2021-2023 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 31 mutual co-author links

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2 shared papersPharmacology & Toxicology

G. Crambert

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Author profile →

2 papers

Recent·Most cited

Low exposure to lithium does not induce nephrogenic diabetes insipidus but microcystic dilations of collecting ducts in a long-term rat model

N. Tabibzadeh, M. Klein, M. Try, J. Poupon, P. Houillier, C. Klein, L. Cheval, G. Crambert, S. Lasaad, L. Chevillard, B. Megarbane

This study shows that low lithium exposure in rats causes microcystic dilations of collecting ducts without inducing nephrogenic diabetes insipidus.

bioRxiv · Oct 12, 2023 · PreprintPharmacology & Toxicology

A new variant of ASIC2 mediates sodium retention in nephrotic syndrome

M. Fila, A. Sassi, G. Brideau, L. Morla, P. Houillier, C. Walter, M. Gennaoui, L. Collignon, M. Keck, G. Planelles, N. Bakouh, M. Peuchmaur, G. Deschenes, I. Anegon, S. Remy, , A. Doucet

The paper identifies a truncated variant of ASIC2b responsible for sodium retention in idiopathic nephrotic syndrome, independent of ENaC and aldosterone.

bioRxiv · Feb 9, 2021Pathology