Author

L.-D. Liao

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cell Biology· 2 Cancer Biology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Research Focus:cancer & tumor microenvironment· 2 Organism:human· 2 Assay:proteomics· 2 Assay:bulk RNA-seq· 1 Research Focus:cell type annotation· 1 Research Focus:chromatin & epigenetic regulation· 1 Research Focus:differential expression· 1

Frequent Co-Authors

E.-M. Li· 3 shared B. Wen· 2 shared L.-Y. Xu· 2 shared C. Li· 1 shared D.-X. Deng· 1 shared G. Neufeld· 1 shared H. Hu· 1 shared H.-X. Zheng· 1 shared

Activity by Year

2022-2024 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 28 mutual co-author links

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3 shared papersCell Biology

E.-M. Li

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

A long non-coding RNA LINC00094 regulates the transcriptional expression of lipid metabolism-related genes as a new member of core regulatory circuitry in esophageal squamous cell carcinoma

L. Peng, Q.-Y. Wang, J.-X. Chen, Y. Chen, R.-Y. Li, L.-D. Liao, W. Lin, C. Li, E.-M. Li, L. Xu

This paper identifies LINC00094 as a long non-coding RNA that forms a core regulatory circuitry with TCF3 and KLF5 to regulate lipid metabolism-related genes in esophageal squamous cell carcinoma.

bioRxiv · Jul 10, 2024 · PreprintCancer Biology

CHAF1A promotes the translesion DNA synthesis pathway in response to DNA replication stress

B. Wen, H.-X. Zheng, D.-X. Deng, Z.-D. Zhang, J.-H. Heng, L.-D. Liao, L.-Y. Xu, E.-M. Li

This study identifies CHAF1A as a novel regulator of the translesion DNA synthesis pathway, enhancing cancer cell survival under DNA replication stress.

bioRxiv · Apr 21, 2023 · PreprintCell Biology

LOXL2-dependent deacetylation of aldolase A induces metabolic reprogramming and tumor progression

J.-W. Jiao, X.-H. Zhan, J.-J. Wang, L.-X. He, Z.-C. Guo, X.-E. Xu, L.-D. Liao, X. Huang, B. Wen, Y.-W. Xu, H. Hu, G. Neufeld, Z.-J. Chang, K. Zhang, L.-Y. Xu, E.-M. Li

This study identifies LOXL2 and its variant L2{Delta}13 as deacetylases that reprogram tumor metabolism by deacetylating aldolase A, promoting tumor progression.

bioRxiv · Mar 6, 2022Cell Biology