Author

M. Armaka

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cell Biology· 2 Cancer Biology· 1 Immunology· 1 Pathology· 1

Signature Assays, Organisms & Methods

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Organism:mouse· 5 Approach:experimental assay· 4 Research Focus:cell type annotation· 3 Assay:ATAC-seq· 1 Assay:bulk RNA-seq· 1 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell signaling & trafficking· 1 Approach:deep learning· 1

Frequent Co-Authors

G. Kollias· 3 shared A. Liakos· 2 shared C. Tzaferis· 2 shared D. Konstantopoulos· 2 shared F. Roumelioti· 2 shared M. Fousteri· 2 shared M. Sakkou· 2 shared N. Karagianni· 2 shared

Activity by Year

2019-2023 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 37 mutual co-author links

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3 shared papersCell Biology

G. Kollias

Mutual co-authors in cluster

Author profile →

5 papers

Recent·Most cited

Cyld aborts hyperactivation of synovial fibroblasts in inflammatory arthritis by regulating the TAK1/IKK2 signaling axis

V. Rinotas, K. Iliaki, L. Pavlidi, T. Meletakos, G. Mosialos, M. Armaka

This study shows that Cyld regulates TNF-induced hyperactivation of synovial fibroblasts in arthritis by controlling the TAK1/IKK2 signaling axis, thereby preventing inflammation and joint destruction.

bioRxiv · Nov 13, 2023Immunology

Single-cell chromatin and transcriptome dynamics of Synovial Fibroblasts transitioning from homeostasis to pathology in modelled TNF-driven arthritis

M. Armaka, D. Konstantopoulos, C. Tzaferis, M. D. Lavigne, M. Sakkou, A. Liakos, P. P. Sfikakis, M. A. Dimopoulos, M. Fousteri, G. Kollias

This study profiles single-cell transcriptomes and chromatin accessibility of synovial fibroblasts in TNF-driven arthritis, identifying disease-specific subtypes and key regulatory factors.

Genome Medicine · Jul 26, 2022Peer-reviewedCell Biology

miR-221/222 drive synovial fibroblast expansion and pathogenesis of TNF-mediated arthritis

F. Roumelioti, C. Tzaferis, D. Konstantopoulos, D. Papadopoulou, A. Prados, M. Sakkou, A. Liakos, P. Chouvardas, T. Meletakos, Y. Pandis, N. Karagianni, M. Denis, M. Fousteri, M. Armaka, G. Kollias

This study shows miR-221/222 promotes synovial fibroblast expansion and worsens TNF-mediated arthritis by regulating cell cycle and ECM pathways.

bioRxiv · Jul 22, 2022 · PreprintCell Biology

Ectopic bone formation and systemic bone loss in a transmembrane TNF-driven model of human spondyloarthritis

E. Christodoulou-Vafeiadou, C. Geka, L. Ntari, K. Kranidioti, E. Argyropoulou, F. Meier, M. Armaka, I. Mourouzis, C. Pantos, M. Rouchota, G. Loudos, M. C. Denis, N. Karagianni, G. Kollias

This paper characterizes a transmembrane TNF-driven mouse model of spondyloarthritis, showing ectopic bone formation, systemic bone loss, and comorbid heart valve pathology, with early anti-TNF treatment reducing inflammation.

bioRxiv · May 14, 2020Pathology

Dual airway and alveolar contributions to adult lung homeostasis and carcinogenesis

M. Spella, I. Lilis, M. A. Pepe, Y. Chen, M. Armaka, A.-S. Lamort, D. E. Zazara, F. Roumelioti, M. Vreka, N. I. Kanellakis, D. E. Wagner, A. D. Giannou, V. Armenis, K. A. M. Arendt, L. V. Klotz, , G. T. Stathopoulos

This study identifies airway contributions to alveolar maintenance and lung adenocarcinoma development through mouse models and transcriptomic analysis.

bioRxiv · Jan 27, 2019Cancer Biology