Author

M. Schlesinger

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 2 Cell Biology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Organism:human· 3 Assay:bulk RNA-seq· 2 Research Focus:cancer & tumor microenvironment· 2 Research Focus:cell type annotation· 2 Assay:proteomics· 2 Research Focus:cell signaling & trafficking· 1 Research Focus:chromatin & epigenetic regulation· 1

Frequent Co-Authors

A. Abu Rmaileh· 1 shared A. Khatib· 1 shared C. Premer· 1 shared D. Salyakina· 1 shared E. Mesri· 1 shared G. Oren· 1 shared H. L. Chan· 1 shared J. H. Axelrod· 1 shared

Activity by Year

2018-2019 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 21 mutual co-author links

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1 shared paperCancer Biology

A. Abu Rmaileh

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

Glutathione Peroxidase 8 (GPX8)-IL6 axis is essential in maintaining breast cancer mesenchymal stem-like state and aggressive phenotype

A. Khatib, S. Balakrishnan, M. Ben-Yosef, G. Oren, A. Abu Rmaileh, M. Schlesinger, J. H. Axelrod, M. Lichtenstein, Y. D. Shaul

This study identifies the GPX8-IL6 axis as a key driver of the mesenchymal stem-like state and aggressive phenotype in breast cancer.

bioRxiv · Oct 25, 2019 · PreprintCancer Biology

PDGFRA Defines the Mesenchymal Stem Cell Kaposi’s Sarcoma Progenitors by Enabling KSHV Oncogenesis in an Angiogenic Environment

J. Naipauer, S. Rosario, S. Gupta, C. Premer, O. Mendez, M. Schlesinger, V. Ponzinibbio, V. Jain, L. Gay, R. Renne, H. L. Chan, L. Morey, D. Salyakina, M. Abba, S. Williams, , E. Mesri

The paper identifies PDGFRA+ bone marrow-derived mesenchymal stem cells as Kaposi’s sarcoma progenitors and shows pro-angiogenic conditions enable KSHV oncogenesis through epigenetic de-repression.

bioRxiv · Oct 2, 2019Cancer Biology

Procyclic Trypanosoma brucei cell cycle is impaired at the G1 stage by the presence of poly(ADP-ribose) in the nucleus

M. Schlesinger, S. C. Vilchez Larrea, S. H. Fernandez Villamil

The paper shows that excess nuclear poly(ADP-ribose) delays the G1 phase of the cell cycle in procyclic Trypanosoma brucei and localizes poly(ADP-ribose) near the mature basal body.

bioRxiv · Oct 25, 2018 · PreprintCell Biology