Author

M. Teh

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 1 Cell Biology· 1 Immunology· 1

Signature Assays, Organisms & Methods

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Assay:bulk RNA-seq· 2 Approach:experimental assay· 2 Organism:human· 2 Research Focus:cell signaling & trafficking· 1 Research Focus:clustering· 1 Research Focus:differential expression· 1 Research Focus:gene regulatory network inference· 1 Research Focus:host-pathogen & immune response· 1

Frequent Co-Authors

A. E. Armitage· 1 shared A. E. Preston· 1 shared A. L. K. Tan· 1 shared A. Nijnik· 1 shared B. Ghesquiere· 1 shared C. Babbs· 1 shared C. Khor· 1 shared D. Higgs· 1 shared

Activity by Year

2019-2023 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 14 mutual co-author links

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1 shared paperCell Biology

A. E. Armitage

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

Ancient genomic linkage couples metabolism with erythroid development

A. E. Preston, J. N. Frost, M. Badat, M. Teh, A. E. Armitage, R. Norfo, S. K. Wideman, M. Hanifi, N. White, N. Roy, B. Ghesquiere, C. Babbs, M. Kassouf, J. Davies, J. Hughes, , H. Drakesmih

This study identifies Nprl3 as a key regulator of erythroid metabolism, showing its role in coupling metabolic and developmental processes during erythropoiesis.

bioRxiv · Sep 25, 2023 · PreprintCell Biology

Spatiotemporal Genomic Profiling of Intestinal Metaplasia Reveals Clonal Dynamics of Gastric Cancer Progression

K. K. Huang, H. Ma, T. Uchihara, T. Sheng, R. H. H. Chong, F. Zhu, S. Srivastava, S. T. Tay, R. Sundar, A. L. K. Tan, X. Ong, M. Lee, S. W. T. Ho, T. Lesluyes, P. Van Loo, , P. Tan

This study analyzes genomic and transcriptomic data from gastric samples to identify driver genes and clonal dynamics in intestinal metaplasia, showing their progression to gastric cancer and the role of microbial dysbiosis.

bioRxiv · Apr 10, 2023Cancer Biology

A loss-of-function mutation in Itgal contributes to the high susceptibility of Collaborative Cross strain CC042 to Salmonella infections

J. Zhang, M. Teh, J. Kim, M. M. Eva, R. Cayrol, R. K. Meade, A. Nijnik, X. Montagutelli, D. Malo, J. Jaubert

This study identifies a loss-of-function mutation in Itgal as a contributor to extreme susceptibility to Salmonella infection in the Collaborative Cross strain CC042.

bioRxiv · Aug 2, 2019 · PreprintImmunology