Author

M. Vieth

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 2

Signature Assays, Organisms & Methods

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Research Focus:cancer & tumor microenvironment· 2 Approach:experimental assay· 2 Organism:mouse· 2 Assay:bulk RNA-seq· 1 Organism:human· 1

Frequent Co-Authors

K. Steiger· 2 shared R. Oellinger· 2 shared R. Rad· 2 shared A. Anand· 1 shared A. Dietl· 1 shared A. Proano-Vasco· 1 shared A. Ralser· 1 shared A. Schnieke· 1 shared

Activity by Year

2024-2024 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 33 mutual co-author links

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2 shared papersCancer Biology

K. Steiger

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2 papers

Recent·Most cited

KRas, in addition to Tp53 is a driver for early carcinogenesis and a molecular target in a mouse model of invasive gastro-esophageal adenocarcinoma

A. Anand, L. Schoemig, S. Lange, L. Tran, K. Flisikowski, R. Oellinger, R. Rad, M. Vieth, K. Steiger, T. Baumeister, J. Strangmann, H. Alguel, K. Ciecielski, K. Boettcher, H.-Y. Fang, , M. Quante

This study identifies KRAS mutation as a driver of invasive gastro-esophageal adenocarcinoma in a mouse model, showing its synergistic role with Tp53 or Rb1 loss and potential as a therapeutic target.

bioRxiv · May 7, 2024 · PreprintCancer Biology

RNF43 is a gatekeeper for colitis-associated cancer

A. Dietl, A. Ralser, K. Taxauer, T. Dregelies, W. Sterlacci, M. Stadler, R. Olayo-Alarcon, S. Margaryan, M. Skerhut, T. Groll, K. Steiger, D. Singh, X. Li, R. Oellinger, R. Rad, , R. Mejias Luque

This study identifies RNF43 mutations as a driver of colitis-associated cancer by showing their role in promoting inflammation and dysbiosis in mouse models and human patients.

bioRxiv · Jan 30, 2024 · PreprintCancer Biology