Author

N. Faumont

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

Click to filter author papers

Cancer Biology· 5 Immunology· 1

Signature Assays, Organisms & Methods

Click to filter · or open tag

Approach:experimental assay· 6 Organism:mouse· 6 Research Focus:host-pathogen & immune response· 3 Research Focus:cancer & tumor microenvironment· 2 Research Focus:cell type annotation· 2 Research Focus:gene regulatory network inference· 2 Assay:bulk RNA-seq· 1 Research Focus:differential expression· 1

Frequent Co-Authors

J. Feuillard· 6 shared C. Vincent-Fabert· 4 shared A. Saintamand· 2 shared C. Ouk· 2 shared F. Boyer· 2 shared L. Roland· 2 shared U. Zimber-Strobl· 2 shared A. David· 1 shared

Activity by Year

2018-2023 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 26 mutual co-author links

Click any collaborator node to inspect or filter shared papers

Focal author (center)Selected co-authorORCID co-authorUnlinked name

Curved chords = mutual co-authorship

6 shared papersCancer Biology

J. Feuillard

Mutual co-authors in cluster

Author profile →

6 papers

Recent·Most cited

REL deregulation stands as a primary hit for AID-imprinted B-cells along the germinal center competition

L. Prevaud, C. Vincent-Fabert, T. Marchiol, Q. Lemasson, C. Ouk, C. Carrion, M. Cogne, J. Feuillard, N. Faumont

This study shows REL deregulation gives AID-imprinted B-cells a competitive advantage in germinal centers, promoting expansion and potential transformation into aggressive lymphomas.

bioRxiv · Oct 10, 2023 · PreprintCancer Biology

Alternative c-MYC mRNA transcripts as an additional tool for c-Myc2 and c-MycS production in BL60 tumors.

D. IBRAHIM, N. FAUMONT, D. TROUTAUD, J. FEUILLARD, M. DIAB-ASSAF, A. OULMOUDEN

The paper identifies five c-MYC transcripts in a mouse model linked to t(8;22) translocation, associated with c-Myc2 and c-MycS production and loss of c-Myc1.

bioRxiv · Feb 18, 2022Cancer Biology

Splice switching oligonucleotide mediated gene knockdown in B cells and plasma cells

L. Delpy, A. Marchalot, J.-M. Lambert, F. Boyer, J. Pollet, J. Cook-Moreau, J. Feuillard, N. Faumont

This paper demonstrates the effectiveness of splice switching oligonucleotides in knocking down c-REL and RELA expression in B cells and plasma cells, with potential therapeutic applications in B-lineage cancers.

bioRxiv · Sep 18, 2020 · PreprintImmunology

B-cell enforced expression of the mouse ortholog of MYD88L265P is responsible for Waldenstrom-like B-cell lymphoma.

C. Ouk, L. Roland, A. Saintamand, N. Gachard, M. Thomas, M. Deveza, N. Faumont, J. Feuillard, C. VINCENT-FABERT

This study shows that enforced expression of MYD88L252P in B-cells leads to a lymphoplasmocytic B-cell lymphoma with a serum IgM peak in mice.

bioRxiv · Oct 6, 2019 · PreprintCancer Biology

Pre-Clinical Blocking of PD-L1 molecule, which expression is down regulated by NF-κB, JAK1/JAK2 and BTK inhibitors, induces regression of activated B-cell lymphoma.

C. Vincent-Fabert, L. Roland, U. Zimber-Strobl, J. Feuillard, N. Faumont

The paper shows that blocking PD-L1 in a mouse model of activated B-cell lymphoma leads to tumor regression, suggesting potential for PD-1/PD-L1 axis therapies combined with tyrosine kinase inhibitors.

bioRxiv · Dec 21, 2018Cancer Biology

Reproducing Transformation of Indolent B-cell Lymphoma by T-cell Immunosuppression of L.CD40 Mice.

C. Vincent-Fabert, A. Saintamand, A. David, M. Alizadeh, F. Boyer, N. Arnaud, U. Zimber-Strobl, J. Feuillard, N. Faumont

The paper shows that T-cell immunosuppression in L.CD40 mice transforms indolent B-cell lymphoma into an aggressive form, highlighting immune surveillance's role in disease progression.

bioRxiv · Nov 24, 2018Cancer Biology