Author

N. Terada

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cell Biology· 2 Immunology· 1 Pharmacology & Toxicology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 4 Research Focus:host-pathogen & immune response· 2 Organism:mouse· 2 Research Focus:cell signaling & trafficking· 1 Research Focus:cell type annotation· 1 Research Focus:cell-cell communication· 1 Research Focus:differential expression· 1 Research Focus:gene regulatory network inference· 1

Frequent Co-Authors

A. Savinov· 1 shared B. N. Newby· 1 shared C. E. Mathews· 1 shared D. Egli· 1 shared D. J. Perry· 1 shared H. Ishii· 1 shared H. Khalil· 1 shared H. Kobayashi· 1 shared

Activity by Year

2018-2024 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 16 mutual co-author links

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1 shared paperImmunology

A. Savinov

Mutual co-authors in cluster

Author profile →

4 papers

Recent·Most cited

Phosphodiesterase-5 inhibition inhibits epithelial ATP release and restores detrusor contractility in rats with type 2 diabetes via an increase in bladder blood flow

T. Kabuto, S. Inamura, H. Kobayashi, X. Zha, K. Nagase, M. Taga, M. Seki, N. Tanaka, Y. Okumura, O. Yokoyama, N. Terada

This study shows that PDE-5 inhibition improves bladder function in T2DM rats by increasing blood flow, reducing ATP release, and modulating inflammatory and growth factor markers.

bioRxiv · Mar 26, 2024 · PreprintPharmacology & Toxicology

Mitochondrial protein C15ORF48 is a stress-independent inducer of autophagy that regulates oxidative stress and autoimmunity

Y. Takakura, M. Machida, N. Terada, Y. Katsumi, S. Kawamura, K. Horie, M. Miyauchi, T. Ishikawa, N. Akiyama, T. Seki, T. Miyao, M. Hayama, R. Endo, H. Ishii, Y. Maruyama, , N. Yamaguchi

This study identifies C15ORF48 as a mitochondrial protein that induces stress-independent autophagy, reducing oxidative stress and autoimmunity in thymic epithelial cells.

bioRxiv · Oct 17, 2023Cell Biology

Use of Induced Pluripotent Stem Cells to Build Isogenic Systems and Investigate Type 1 Diabetes

L. H. Armitage, S. E. Stimpson, K. E. Santostefano, L. Sui, S. O. Ogundare, B. N. Newby, R. Castro-Gutierrez, M. K. Huber, J. P. Taylor, P. Sharma, I. Radichev, D. J. Perry, N. Fredette, A. Savinov, N. Terada, , C. E. Mathews

This paper demonstrates the use of induced pluripotent stem cells to create an isogenic system for studying genetic influences on cell-cell interactions in type 1 diabetes.

bioRxiv · May 3, 2021Immunology

Inhibition of mitochondrial permeability transition by deletion of the ANT family and CypD

J. Karch, M. J. Bround, H. Khalil, M. A. Sargent, N. Latchman, N. Terada, P. M. Peixoto, J. D. Molkentin

The paper shows that genetic deletion of Ant1/2/4 and Ppif in mice fully inhibits the mitochondrial permeability transition pore.

bioRxiv · Dec 27, 2018Cell Biology