Author

P. Chakrabarti

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Biochemistry· 1 Cancer Biology· 1 Cell Biology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Organism:human· 3 Assay:bulk RNA-seq· 2 Research Focus:cell signaling & trafficking· 2 Research Focus:host-pathogen & immune response· 2 Research Focus:metabolism & bioenergetics· 1 Assay:proteomics· 1

Frequent Co-Authors

S. Basu· 2 shared S. N. Bhattacharyya· 2 shared A. Das· 1 shared B. k. Pathak· 1 shared D. Bandopadhyay· 1 shared D. Bandyopadhyay· 1 shared D. Das· 1 shared D. Datta· 1 shared

Activity by Year

2019-2021 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 18 mutual co-author links

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2 shared papersCancer Biology

S. Basu

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

Cancer BiologybioRxivJan 4, 2021

Inhibition of Extracellular Vesicle-Associated MMP2 Abrogates Intercellular Transfer of Hepatic miR-122 to Tissue Macrophages and Curtails Liver Inflammation

A. Das, S. Basu, D. Bandyopadhyay…·S. N. Bhattacharyya

The paper shows that inhibiting MMP2 on extracellular vesicles reduces miR-122 transfer to macrophages, thereby decreasing liver inflammation.

DOI 10.1016/j.isci.2021.103428

humanbulk RNA-seq

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Cell BiologybioRxivPreprintNov 30, 2020

Lipid Droplets Promote Phase Separation of Ago2 to Accelerate Dicer1 Loss and Decelerate miRNA Activity in Lipid Exposed Hepatic Cells

D. Bandopadhyay, S. Basu, I. Mukherjee…·S. N. Bhattacharyya

The paper shows lipid droplets promote Ago2 phase separation, reduce Dicer1 levels, and decrease miR-122 activity in hepatic cells under lipotoxic stress.

DOI 10.1101/2020.11.30.405449

humanbulk RNA-seq

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BiochemistrybioRxivPreprintApr 17, 2019

Resveratrol prevents amyloid fibrillation of insulin by arresting it in a bioactive oligomeric form

B. k. Pathak, D. Das, S. Bhakta…·J. Sengupta

This study shows resveratrol prevents insulin fibrillation by keeping it in a bioactive oligomeric form and retains its biological activity without toxicity.

DOI 10.1101/611376

humanproteomics

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