Author

P. Nag

Primary ORCID profile (0009-0000-8923-9189 · 1 paper)Unclaimed ORCID profile · follow to get notified of new preprints

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Same name, identified by ORCID

P. NagiD1 paper · The University of Osaka

Biology Disciplines

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Cancer Biology· 2

Signature Assays, Organisms & Methods

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Research Focus:cancer & tumor microenvironment· 2 Approach:experimental assay· 2 Assay:proteomics· 2 Research Focus:cell signaling & trafficking· 1 Assay:DNA methylation· 1 Research Focus:drug response / drug discovery· 1 Organism:human· 1 Organism:mouse· 1

Frequent Co-Authors

K. K. Khanna· 2 shared A. Burgess· 1 shared A. L. Bain· 1 shared A. S. Trigos· 1 shared C. Cullinane· 1 shared C. L. Scott· 1 shared D. Frank· 1 shared D. Nanayakkara· 1 shared

Activity by Year

2019-2019 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 25 mutual co-author links

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2 shared papersCancer Biology

K. K. Khanna

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2 papers

Recent·Most cited

Cep55 overexpression promotes genomic instability and tumorigenesis in mice

D. Sinha, P. Nag, D. Nanayakkara, P. H. G. Duijf, A. Burgess, P. Raninga, V. A. J. Smits, A. L. Bain, G. N. Subramanian, M. Wall, J. W. Finnie, M. Kalimutho, K. K. Khanna

This study shows that Cep55 overexpression causes genomic instability and tumorigenesis in mice by promoting mitotic errors and disrupting DNA repair mechanisms.

bioRxiv · Sep 24, 2019Cancer Biology

Inhibition of RNA Polymerase I Transcription Activates Targeted DNA Damage Response and Enhances the Efficacy of PARP Inhibitors in High-Grade Serous Ovarian Cancer

E. Sanij, K. M. Hannan, S. Yan, X. Xuan, J. E. Ahern, K. T. Chan, J. Son, O. Kondrashova, E. Lieschke, M. J. Wakefield, A. S. Trigos, D. Frank, S. Ellis, C. Cullinane, J. Kang, , R. B. Pearson

This study shows CX-5461, an RNA polymerase I inhibitor, enhances PARP inhibitor efficacy in HR-deficient HGSOC by inducing replication stress and DNA damage, overcoming resistance mechanisms.

bioRxiv · Apr 29, 2019 · PreprintCancer Biology