Author

R. Ran

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 1 Cell Biology· 1 Immunology· 1

Signature Assays, Organisms & Methods

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Organism:human· 3 Research Focus:cell type annotation· 2 Assay:scRNA-seq· 2 Approach:statistical model· 2 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell signaling & trafficking· 1 Research Focus:cell-cell communication· 1 Research Focus:development & morphogenesis· 1

Frequent Co-Authors

D. Brubaker· 2 shared A. Fowles· 1 shared A. Mastro· 1 shared H. Harrison· 1 shared H. Pegg· 1 shared I. Holen· 1 shared J. Munoz· 1 shared K. Blythe· 1 shared

Activity by Year

2019-2024 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 19 mutual co-author links

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2 shared papersImmunology

D. Brubaker

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

Systematic Analysis of Human Colorectal Cancer scRNA-seq Revealed Limited Pro-tumoral IL-17 Production Potential in Gamma Delta T Cells

R. Ran, M. Trapecar, D. Brubaker

This study analyzes scRNA-seq data from human colorectal cancer to show that gamma delta T cells do not produce pro-tumoral IL-17, functioning mainly as cytotoxic effector cells.

bioRxiv · Jul 18, 2024Immunology

Detailed Survey of an in-vitro Intestinal Epithelium Model by Single-Cell Transcriptomics

R. Ran, J. Munoz, S. Jena, L. Green, D. Brubaker

This study uses single-cell RNA sequencing to analyze a Caco-2/HT29 co-culture model, identifying cell types and assessing its similarity to human intestinal epithelium, finding it lacks some transporters and immune markers.

iScience · Apr 1, 2024Peer-reviewedCell Biology

RUNX/CBFβ transcription factor complexes promote the phenotypic plasticity of metastatic breast cancer cells

R. Ran, H. Harrison, N. S. Ariffin, R. Ayub, H. Pegg, W. Deng, A. Mastro, P. Ottewell, A. Fowles, S. Mason, K. Blythe, I. Holen, P. Shore

This study shows that RUNX/CBFβ complexes drive phenotypic plasticity in metastatic breast cancer cells by promoting EMT and enabling MET reversal.

bioRxiv · Feb 27, 2019 · PreprintCancer Biology