Author

S. Cheon

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 1 Cell Biology· 1 Immunology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Assay:bulk RNA-seq· 2 Organism:human· 2 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell type annotation· 1 Assay:ChIP-seq· 1 Research Focus:chromatin & epigenetic regulation· 1 Research Focus:differential expression· 1

Frequent Co-Authors

C. Park· 2 shared J. Lee· 2 shared C. Arrowsmith· 1 shared C. H. Lee· 1 shared C. Moon· 1 shared D. Barsyte-Lovejoy· 1 shared E. C. Adamson· 1 shared E. Cho· 1 shared

Activity by Year

2020-2023 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 18 mutual co-author links

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2 shared papersCell Biology

C. Park

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

Epigenetic vulnerabilities of leukemia harboring inactivating EZH2 mutations

M. Alqazzaz, G. Luciani, V. Vu, R. M. Machado, M. M. Szewczyk, E. C. Adamson, S. Cheon, C. Arrowsmith, M. D. Minden, D. Barsyte-Lovejoy

This study identifies epigenetic vulnerabilities in AML and MDS/MPN with EZH2 mutations, showing increased sensitivity to EZH2 and EED inhibitors and upregulation of cell death pathways.

bioRxiv · Nov 20, 2023Cancer Biology

Peroxiredoxin 5 regulates osteogenic differentiation via interaction with hnRNPK during bone regeneration

E. Cho, X. Che, M. J. Ang, S. Cheon, J. Lee, K. Kim, C. H. Lee, S.-Y. Lee, H.-Y. Yang, C. Moon, C. Park, J.-Y. Choi, T.-H. Lee

This study identifies Prdx5's role in bone regeneration by regulating osteoblast differentiation through interaction with hnRNPK, impacting osteocalcin expression and osteoclast activity.

bioRxiv · Jun 9, 2022Cell Biology

COVID-19 patients upregulate toll-like receptor 4-mediated inflammatory signaling that mimics bacterial sepsis

K. M. Sohn, S.-G. Lee, H. J. Kim, S. Cheon, H. Jeong, J. Lee, I. S. Kim, P. Silwal, Y. J. Kim, C. Park, Y.-S. Kim, E.-K. Jo

The paper identifies upregulated TLR4-mediated inflammatory signaling in COVID-19 patients, linking it to pathological inflammation and suggesting TLR4 as a therapeutic target.

bioRxiv · Jul 17, 2020Immunology