S. LeibundGut-Landmann

This study shows that impaired skin barriers in atopic dermatitis allow Malassezia yeast to grow more efficiently due to changes in the skin's lipid environment.

This study identifies Vγ4+ γδ T cells as key mediators of IL-17-dependent fungal control against Malassezia in skin, showing memory-like responses and direct recognition of fungal ligands.

This study identifies factors enabling Candida albicans to maintain a commensal lifestyle in the oral mucosa by reducing virulence and adapting metabolically.

This study identifies a novel mycovirus in Malassezia that alters transcriptional profiles and induces interferon-β in macrophages, suggesting potential implications for fungal pathogenicity.

This study identifies bacterially-derived flavohemoglobin in Malassezia, acquired via horizontal gene transfer, as essential for nitric oxide resistance and host interaction.