Author

S. Marcone

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 1 Cell Biology· 1 Immunology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Assay:proteomics· 3 Research Focus:cell type annotation· 2 Organism:human· 2 Research Focus:host-pathogen & immune response· 1 Organism:zebrafish· 1

Frequent Co-Authors

A. Joshi· 1 shared B. N. Kennedy· 1 shared C. Godson· 1 shared D. C. Zeldin· 1 shared D. Cooper· 1 shared D. Tobin· 1 shared E. Brennan· 1 shared H. E. Allan· 1 shared

Activity by Year

2019-2021 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 12 mutual co-author links

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1 shared paperCell Biology

A. Joshi

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

Inhibition of Metastatic Uveal Melanoma Cell Proliferation by the Histone Deacetylase 6 Inhibitor, Ricolinostat, is Linked to Altered Microphthalmia-associated Transcription Factor and Phospho-ERK Expression

H. Sundaramurthi, S. Garcia-Mulero, K. Slater, S. Marcone, J. M. Piulats, R. W. Watson, D. Tobin, L. Jensen, B. N. Kennedy

This study shows that ACY-1215 inhibits metastatic uveal melanoma cell growth in vitro and in vivo by reducing p-ERK and MITF signaling.

bioRxiv · Oct 28, 2021 · PreprintCancer Biology

Proteome and functional decline as platelets age in the circulation

H. E. Allan, M. A. Hayman, S. Marcone, M. V. Chan, M. L. Edin, T. Maffucci, A. Joshi, L. Menke, M. Crescente, M. Mayr, D. C. Zeldin, P. C. Armstrong, T. D. Warner

The paper investigates age-related changes in platelets, finding reduced protein content, altered proteomic pathways, and functional decline in older platelets.

bioRxiv · Jun 2, 2021Cell Biology

Extracellular vesicles from monocyte/platelet aggregates modulate human atherosclerotic plaque responses

S. Oggero, M. de Gaetano, S. Marcone, M. Barry, T. Montero-Melendez, D. Cooper, L. V. Norling, E. Brennan, C. Godson, M. Perretti

This study shows that EVs from monocyte/platelet aggregates modulate atherosclerotic plaque responses, with Iloprost reducing their pro-inflammatory effects.

bioRxiv · Nov 13, 2019 · PreprintImmunology