Author

S. Mathas

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 2

Signature Assays, Organisms & Methods

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Research Focus:cell type annotation· 2 Research Focus:differential expression· 2 Approach:experimental assay· 2 Research Focus:gene regulatory network inference· 2 Organism:mouse· 2 Assay:bulk RNA-seq· 1

Frequent Co-Authors

A. Aufinger· 1 shared A. Heuser· 1 shared A. Rosenwald· 1 shared B. Strobl· 1 shared B. Tichy· 1 shared B. Walter· 1 shared B. Wollert-Wulf· 1 shared C. Farre Diaz· 1 shared

Activity by Year

2021-2023 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 20 mutual co-author links

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1 shared paperCancer Biology

A. Aufinger

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2 papers

Recent·Most cited

JUN mediates senescence and immune cell recruitment to prevent prostate cancer progression

T. Redmer, M. Raigel, C. Sternberg, R. Ziegler, C. Probst, D. Lindner, A. Aufinger, T. Limberger, K. Trachtova, P. Kodajova, S. Hoegler, M. Schlederer, S. Stoiber, M. Oberhuber, M. Bolis, , L. Kenner

This study shows JUN suppresses prostate cancer progression by inducing senescence and immune cell recruitment, with JUN depletion accelerating tumor growth.

bioRxiv · Nov 29, 2023 · PreprintCancer Biology

Preclinical models of human multiple myeloma subgroups

W. Winkler, C. Farre Diaz, E. Blanc, H. Napieczynska, P. Langner, M. Werner, B. Walter, B. Wollert-Wulf, T. Yasuda, A. Heuser, D. Beule, S. Mathas, I. Anagnostopoulos, A. Rosenwald, K. Rajewsky, M. Janz

This study generates preclinical models of human multiple myeloma subgroups by inducing specific genetic alterations in mice, showing their resemblance to human disease.

bioRxiv · Aug 28, 2021 · PreprintCancer Biology