Author

T. Bogenrieder

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 2

Signature Assays, Organisms & Methods

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Research Focus:cancer & tumor microenvironment· 2 Approach:experimental assay· 2 Assay:DNA methylation· 1 Research Focus:drug response / drug discovery· 1 Organism:human· 1 Organism:mouse· 1 Assay:proteomics· 1

Frequent Co-Authors

U. Weyer-czernilofsky· 2 shared A. J. Ryan· 1 shared A. Mielgo· 1 shared A. Nandakumar· 1 shared A. Santos· 1 shared C. Figueiredo· 1 shared D. Ebner· 1 shared E. Seraia· 1 shared

Activity by Year

2017-2020 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 25 mutual co-author links

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2 shared papersCancer Biology

U. Weyer-czernilofsky

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2 papers

Recent·Most cited

IGF targeting perturbs global replication through ribonucleotide reductase dysfunction

G. Rieunier, X. Wu, L. Harris, J. Mills, A. Nandakumar, L. Colling, E. Seraia, S. B. Hatch, D. Ebner, L. Folkes, U. Weyer-czernilofsky, T. Bogenrieder, A. J. Ryan, V. M. Macaulay

This study shows that IGF-1R inhibition causes replication stress by reducing RNR subunit RRM2, leading to DNA damage and synthetic lethality with ATM loss in cancer cells.

bioRxiv · Aug 11, 2020Cancer Biology

Blockade of insulin-like growth factors increases efficacy of paclitaxel in metastatic breast cancer.

L. Ireland, A. Santos, F. Campbell, C. Figueiredo, L. Ellies, U. Weyer-Czernilofsky, T. Bogenrieder, M. Schmid, A. Mielgo

This study shows that blocking insulin-like growth factors enhances paclitaxel's effectiveness against metastatic breast cancer by reducing tumor growth and lung metastasis in pre-clinical models.

bioRxiv · Jul 23, 2017Cancer Biology