Author

T. C. Moller

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Biophysics· 1 Pharmacology & Toxicology· 1

Signature Assays, Organisms & Methods

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Research Focus:cell signaling & trafficking· 1 Research Focus:cell type annotation· 1 Assay:fluorescence / live-cell microscopy· 1 Approach:genetic perturbation / CRISPR· 1 Organism:human· 1 Approach:microscopy & imaging analysis· 1

Frequent Co-Authors

E. Margeat· 1 shared E. Trinquet· 1 shared H. Bräuner-Osborne· 1 shared H. Liu· 1 shared J. Liu· 1 shared J. M. Mathiesen· 1 shared J. R. van Senten· 1 shared J.-P. Pin· 1 shared

Activity by Year

2020-2022 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 21 mutual co-author links

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1 shared paperBiophysics

E. Margeat

Mutual co-authors in cluster

Author profile →

2 papers

Recent·Most cited

BiophysicsbioRxivPreprintJan 7, 2022

Dissecting conformational rearrangements and allosteric modulation in metabotropic glutamate receptor activation

N. Lecat-Guillet, R. B. Quast, H. Liu…·E. Margeat

The paper uses FRET sensors to study conformational changes in mGlu2 receptors, showing ligands modulate activation through multiple states.

DOI 10.1101/2022.01.07.474531

humanfluorescence / live-cell microscopy

𝕏 PostBluesky

Pharmacology & ToxicologybioRxivJan 8, 2020

Dissecting the roles of GRK2 and GRK3 in μ-opioid receptor internalization and β-arrestin2 recruitment using CRISPR/Cas9-edited HEK293 cells

T. C. Moller, M. F. Pedersen, J. R. van Senten…·H. Bräuner-Osborne

This study uses CRISPR/Cas9 to knockout GRK2 and GRK3 in HEK293 cells to assess their roles in μ-opioid receptor internalization and β-arrestin2 recruitment, finding GRK2 more critical than GRK3.

DOI 10.1038/s41598-020-73674-0

genetic perturbation / CRISPRcell type annotation

𝕏 PostBluesky