T. Gil-Gil

The paper investigates phage resistance in E. coli due to O-antigen expression and finds that most E. coli in the gut are resistant, limiting phage impact on bacterial composition.

This study shows that long-term exposure to bacteriostatic antibiotics in E. coli leads to the emergence of small colony variants (SCVs) that confer heteroresistance, which reverts upon antibiotic removal.

This paper develops two mathematical models of heteroresistance to explore how resistance ascends and reverts during antibiotic treatment, showing that current identification methods are insufficient.

This study investigates how nutrient conditions affect antibiotic pharmacodynamics by comparing MICs and bacterial growth in rich and minimal media with E. coli and eight antibiotics.

This paper identifies a novel mechanism of antibiotic resistance in Stenotrophomonas maltophilia through the inactivation of central carbon metabolism enzymes, not through known resistance mechanisms like efflux or target modification.