JinSeok Park

This paper uses machine learning to infer neuroblastoma cell states from morphology without transcriptomics, showing alignment with scRNA-seq and tracking state transitions.

This study identifies a YAP-PIEZO1 signaling axis that links extracellular matrix density to invasion in fusion-negative rhabdomyosarcoma, with therapeutic implications.

This study shows that ECM topography promotes adrenergic to mesenchymal transition in neuroblastoma by enhancing cell-ECM interactions and activating the Rho-associated kinase pathway.

This study identifies YAP's role in controlling cell migration and invasion via a Rho-GTPase switch, with implications for glioblastoma progression.

This study identifies differences in cell-ECM interaction between rhabdomyosarcoma subtypes, showing FNRMS has heightened interaction regulated by TGFβ signaling, while PAX3-FOXO1 in FPRMS suppresses this interaction.