Julie C. Canman

This study identifies germ lineage-specific DAF-16 patterning and a role for DAF-16 in early embryogenesis independent of canonical DAF-2 signaling.

This study shows that time-restricted eating in Drosophila leads to sustained fat loss and improved muscle function without calorie restriction.

This study shows that evolving the Drosophila gut microbiome to resist oxidative stress improves host health and extends lifespan.

The paper compares microtubule dynamics in C. elegans embryos using fluorescent labels and finds high variability, with spindle assembly robustness despite dynamic changes and potential issues with tagged end-binding proteins.

The paper identifies a novel anaphase A mechanism in C. elegans embryos driven by KLP-7MCAK and opposed by KLP-18, showing its role in asymmetric chromosome segregation and mechanical coordination with anaphase B.

This study investigates how chronic short sleep affects metabolism and survival under oxidative stress in mutants, finding common metabolic defects and suggesting lipid metabolism issues may underlie oxidative stress sensitivity.

The paper identifies an interkinetic envelope surrounding chromosomes in C. elegans oocytes during interkinesis, showing it is a distinct structure from the nuclear envelope.

The paper identifies cell type-specific differences in cytokinesis regulation in C. elegans embryos using temperature-sensitive mutants.

This study investigates the spatial and temporal dynamics of FoxO/DAF-16 in C. elegans embryos and its role in cell cycle regulation during early development.

The paper identifies PIE-1 and POS-1 as germ fate determinants that protect P2 germ precursor cells from cytokinesis failure by reducing septin and anillin levels at the division plane.

The paper uses CRISPR to disrupt clock genes in specific Drosophila neurons, showing that neither the morning nor evening oscillator alone is sufficient for circadian locomotor activity, indicating a distributed network function.