Takehiro Suzuki

This study reports cryo-EM structures of BAM-BepA complexes and identifies loop conformations that regulate BepA's dual function in substrate triage.

This paper identifies CgLhcf9 as a negative regulator of energy-dependent NPQ in the diatom Chaetoceros gracilis, showing it suppresses NPQ under specific light and CO2 conditions to improve growth.

This study presents the cryo-EM structure of TOK1, revealing its unique domain architecture and lipid-mediated gating mechanism through structural and functional analyses.

This study investigates the subcellular localization of Bcnt/Cfdp1, an intrinsically disordered protein, revealing its presence in both nucleus and cytoplasm, with mobility affected by macromolecular crowding.

The paper elucidates the cleavage cascade of FecR that mediates ferric citrate signaling via the TonB-FecA axis in Escherichia coli.

This study identifies IL-1β-inducing pathobionts in Crohn's disease that drive inflammation and may predict response to anti-TNF therapies.

The paper identifies a plant immune receptor, SCORE, that recognizes CSP peptides and shows polymorphisms across species, enabling the design of synthetic variants for broad pathogen detection.

The paper identifies a novel chlorophyll a/b-binding protein, RedCAP, in the PSI-LHCI supercomplexes of Galdieria sulphuraria, highlighting its distinct binding properties and spectral characteristics compared to LHC proteins.

The paper determines the structure of PSI-LHCI from Cyanidium caldarium and investigates its evolutionary relationships with other red-lineage LHCs.

This study investigates the effects of eIF5A hypusination inhibition on mitochondrial function and identifies MPV17L2 as a key regulator involved in mitochondrial integrity.

This study identifies METTL18 as a histidine methyltransferase that modifies RPL3, slowing ribosome translocation on tyrosine codons to aid protein folding and prevent aggregation.

The paper examines changes in PSD protein composition across development in mice and primates, linking these changes to autism spectrum disorder.

The paper shows splicing modulators cause translational repression by inducing condensate-prone intron-derived proteins that inhibit mTORC1 and trigger proteotoxic stress.

The paper investigates the role of H3K9me2 in 3D genome organization by analyzing its distribution, transcriptome, and chromatin structure in mouse embryonic stem cells with altered H3K9 methyltransferase activity.