Author

Wen Dun

Columbia UniversityORCID 0000-0002-3687-8021Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Organism:mouse· 2 Research Focus:cell signaling & trafficking· 1 Research Focus:host-pathogen & immune response· 1 Organism:human· 1 Assay:imaging· 1 Organism:rat· 1

Frequent Co-Authors

Akinola Alafiatayo· 1 shared Bruno D. M. Y. StuyversiD· 1 shared Bruno QuessoniD· 1 shared Christos TzimasiD· 1 shared Emily J. TsaiiD· 1 shared Fabien BretteiD· 1 shared Henk E.D.J. ter Keurs· 1 shared Ilaria RussoiD· 1 shared

Activity by Year

2023-2024 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 24 mutual co-author links

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1 shared paperCell Biology

Akinola Alafiatayo

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2 papers

Recent·Most cited

PhysiologyAmerican Journal of Physiology-Heart and Circulatory PhysiologyPeer-reviewedDec 16, 2024

Extracellular Matrix Instability and Chronic Inflammation Underlie Maladaptive Right Ventricular Pressure Overload Remodeling and Failure in Male Mice

Ilaria Russo, Wen Dun, Swasti Mehta…·Emily J. Tsai

This study shows male mice with pulmonary artery banding develop severe right ventricular dysfunction and failure due to ECM instability and chronic inflammation, while females show more adaptive remodeling without these features.

10 citations

mouseexperimental assay

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Cell BiologybioRxivPreprintOct 23, 2023

Increased spontaneous Ca2+ activity in Cardiac Purkinje cells after myocardial infarction; A consequence of a dramatic shift of SERCA isoforms as potential adaptation to acute ischemia?

Bruno D. M. Y. Stuyvers, Penelope A. Boyden, Ruhul Amin…·Touati Benoukraf

This study shows that increased spontaneous Ca2+ activity in cardiac Purkinje cells after myocardial infarction is due to a shift in SERCA2 isoforms, specifically increased SERCA2b and reduced SERCA2a.

DOI 10.1101/2023.10.23.563678

humanmouse

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