Author

Danielle A. Garsin

The University of Texas Health Science Center at HoustonORCID 0000-0002-6551-2600Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

Click to filter author papers

Microbiology· 2 Genetics· 1 Molecular Biology· 1

Signature Assays, Organisms & Methods

Click to filter · or open tag

Approach:experimental assay· 3 Research Focus:host-pathogen & immune response· 2 Organism:human· 2 Organism:bacteria (other)· 1 Organism:C. elegans· 1 Research Focus:drug response / drug discovery· 1 Approach:genetic perturbation / CRISPR· 1 Organism:mouse· 1

Frequent Co-Authors

Melissa R. CruziD· 3 shared A. Q. DinhiD· 1 shared Ambro van HoofiD· 1 shared April H. NguyeniD· 1 shared Ayan ChatterjeeiD· 1 shared Ayesha KhaniD· 1 shared Bhawana IsraniiD· 1 shared Carolaing Gabaldón· 1 shared

Activity by Year

2024-2026 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 17 mutual co-author links

Click any collaborator node to inspect or filter shared papers

Focal author (center)Selected co-authorORCID co-authorUnlinked name

Curved chords = mutual co-authorship

3 shared papersMolecular Biology

Melissa R. CruzORCID

Mutual co-authors in cluster

Author profile →

4 papers

Recent·Most cited

Molecular BiologybioRxivPreprintAug 20, 2026

Characterization of tRNA ligase function in pathogenic fungi and trypanosomes reveals the ligase domain as a promising drug target

Khondakar Sayef Ahammed, Pedro Miramón, Lukas Schrettenbrunner…·Ambro van Hoof

The paper identifies the Trl1 sealing domain as a promising drug target for antifungal and anti-trypanosomal therapies by analyzing its essentiality in pathogenic fungi and trypanosomes.

DOI 10.64898/2026.08.16.745097

humanmouse

𝕏 PostBluesky

GeneticsbioRxivPreprintJul 26, 2026

LIN-23 Affects C. elegans Pathogen and Stress Resistance by Modulating SKN-1 Activity

Larissa A Tavizon, Carolaing Gabaldón, Melissa R. Cruz…·Danielle A. Garsin

This study identifies LIN-23 as a positive regulator of SKN-1 activity in C. elegans during pathogen exposure, showing it modulates WDR-23 to enhance stress resistance.

DOI 10.64898/2026.07.22.740106

C. elegansexperimental assay

𝕏 PostBluesky

MicrobiologyInfection and ImmunityPeer-reviewedMay 16, 2024

Role of Ethanolamine Utilization and Bacterial Microcompartment Formation in Listeria monocytogenes Intracellular Infection

Ayan Chatterjee, Karan Gautam Kaval, Danielle A. Garsin

This study investigates the role of ethanolamine utilization and bacterial microcompartment formation in Listeria monocytogenes intracellular infection, finding that EA utilization mutants show similar replication defects and immune responses as BMC formation mutants.

6 citations

humanproteomics

𝕏 PostBluesky

MicrobiologyThe Journal of Infectious DiseasesPeer-reviewedApr 3, 2024

Membrane Lipids Augment Cell Envelope Stress Signaling and Resistance to Antibiotics and Antimicrobial Peptides in Enterococcus faecalis

William R. Miller, April H. Nguyen, Kavindra V. Singh…·César A. Arias

This study identifies the MadRS system and its operon madEFG as mediators of resistance to antimicrobial peptides and antibiotics in Enterococcus faecalis, linking membrane lipid changes to stress response and virulence.

16 citations

bacteria (other)genetic perturbation / CRISPR

𝕏 PostBluesky