Author

Callum J Quinn

University of ManchesterORCID 0000-0003-1713-5975Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Signature Assays, Organisms & Methods

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Research Focus:development & morphogenesis· 2 Approach:experimental assay· 2 Research Focus:cell signaling & trafficking· 1 Research Focus:cell type annotation· 1 Assay:electrophysiology· 1 Organism:mouse· 1

Frequent Co-Authors

Andrew W. TraffordiD· 2 shared Katharine M. DibbiD· 2 shared Alice S. WhitleyiD· 1 shared C. Booth· 1 shared Charlotte E. R. SmithiD· 1 shared Christian PinaliiD· 1 shared David A. EisneriD· 1 shared David Charles HutchingsiD· 1 shared

Activity by Year

2025-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 30 mutual co-author links

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2 shared papersPhysiology

Andrew W. TraffordORCID

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2 papers

Recent·Most cited

PhysiologyJournal of Molecular and Cellular CardiologyPeer-reviewedJan 4, 2026

Atrial t-tubules adopt a specialist developmental state while alterations to Ca2+ buffering maintain systolic Ca2+ during postnatal development

Charlotte E. R. Smith, Callum J Quinn, Jessica D. Clarke…·Katharine M. Dibb

This study examines atrial t-tubule development in sheep, showing that despite reduced density in newborns, Ca2+ handling is maintained through increased L-type Ca2+ current and sarcoplasmic reticulum content.

2 citationstop 5%

electrophysiologyexperimental assay

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PhysiologybioRxivPreprintAug 20, 2025

Dysferlin Regulates Cardiac T-tubule Structure and Excitation-contraction Coupling in Isolated Cardiac Myocytes at Rest and in Response to Acute Hypo-osmotic Stress and is Protective Against Arrhythmias in Langendorff-perfused Hearts

Callum J Quinn, C. Booth, Kerri L. M. Smith…·Katharine M. Dibb

This study shows dysferlin is critical for maintaining cardiac T-tubule structure and EC coupling, and its loss increases arrhythmia risk under stress and during aging.

DOI 10.1101/2025.08.20.671306

mouseexperimental assay

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