Dhananjay D. Shinde

This study identifies a thermodynamic bottleneck in the TCA cycle of S. aureus that favors acetate overflow metabolism for bioenergetic efficiency.

This study shows that Toxoplasma gondii exploits host ER-phagy to increase amino acid levels, which supports its persistence and reduces neurological effects in mice.

This study shows that Staphylococcus aureus counters acetate-mediated inhibition of peptidoglycan cross-linking by maintaining high intracellular D-Ala levels through alanine racemase and limiting D-Ala flux via D-alanine aminotransferase.