Author

A. Chalk

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 2

Signature Assays, Organisms & Methods

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Research Focus:cancer & tumor microenvironment· 2 Approach:experimental assay· 2 Organism:mouse· 2 Research Focus:cell type annotation· 1

Frequent Co-Authors

C. Walkley· 2 shared A. G. Kulasekararaj· 1 shared A. Goradia· 1 shared A. Unnikrishnan· 1 shared C. Grace· 1 shared C. Joseph· 1 shared D. J. McCarthy· 1 shared E. Hellstrom-Lindberg· 1 shared

Activity by Year

2023-2023 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 37 mutual co-author links

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2 shared papersCancer Biology

C. Walkley

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2 papers

Recent·Most cited

Dysregulated expression of Hoxa1 isoforms in hematopoietic stem and progenitor cells causes myelodysplastic syndromes.

S. Y. Tan, C. Joseph, M. L. Sobah, A. Chalk, K. Schleibs, S. Lee, G. Tjin, S. Chugh, X. Mo, C. Grace, J. Q. Truong, M. F. Smeets, K. Rice, M. Parker, A. Unnikrishnan, , L. Purton

This study shows that dysregulated Hoxa1 isoforms in hematopoietic stem and progenitor cells cause myelodysplastic syndromes and provide mouse models for disease progression.

bioRxiv · Apr 24, 2023 · PreprintCancer Biology

Over-expression of ADAR1 in mice does not initiate or accelerate cancer formation in vivo.

S. Mendez Ruiz, A. Chalk, A. Goradia, J. Heraud-Farlow, C. Walkley

This study shows that overexpressing ADAR1 in mice does not cause or speed up cancer formation, suggesting ADAR1 elevation is a passive, not active, factor in tumors.

bioRxiv · Jan 29, 2023 · PreprintCancer Biology