Author

C. Joseph

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

Click to filter author papers

Cell Biology· 2 Cancer Biology· 1 Immunology· 1

Signature Assays, Organisms & Methods

Click to filter · or open tag

Approach:experimental assay· 3 Organism:human· 3 Research Focus:cell type annotation· 2 Research Focus:differential expression· 2 Assay:proteomics· 2 Assay:scRNA-seq· 2 Assay:bulk RNA-seq· 1 Research Focus:cancer & tumor microenvironment· 1

Frequent Co-Authors

A. Banerjee· 1 shared A. C. Doxey· 1 shared A. C. Habermann· 1 shared A. CAMPOS· 1 shared A. Chalk· 1 shared A. Chandiramohan· 1 shared A. Dvorkin-Gheva· 1 shared A. G. Kulasekararaj· 1 shared

Activity by Year

2020-2023 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 10 mutual co-author links

Click any collaborator node to inspect or filter shared papers

Focal author (center)Selected co-authorORCID co-authorUnlinked name

Curved chords = mutual co-authorship

1 shared paperImmunology

A. Banerjee

Mutual co-authors in cluster

Author profile →

4 papers

Recent·Most cited

Regulation of Non-Canonical Proteins Encoded by Small Open Reading Frames via the Nonsense-Mediated Decay Pathway.

P. PERIASAMY, C. JOSEPH, A. CAMPOS, S. RAJANDRAN, C. Batho, J. E. Hudson, H. Sivakumaran, H. Kore, K. Datta, J. Yeong, H. Gowda

This study investigates how UPF1 knockdown affects non-canonical/mutant protein expression and reveals its role in suppressing neoantigen availability through the NMD pathway in cancer.

bioRxiv · Aug 26, 2023 · PreprintCell Biology

Dysregulated expression of Hoxa1 isoforms in hematopoietic stem and progenitor cells causes myelodysplastic syndromes.

S. Y. Tan, C. Joseph, M. L. Sobah, A. Chalk, K. Schleibs, S. Lee, G. Tjin, S. Chugh, X. Mo, C. Grace, J. Q. Truong, M. F. Smeets, K. Rice, M. Parker, A. Unnikrishnan, , L. Purton

This study shows that dysregulated Hoxa1 isoforms in hematopoietic stem and progenitor cells cause myelodysplastic syndromes and provide mouse models for disease progression.

bioRxiv · Apr 24, 2023 · PreprintCancer Biology

Chronic lung diseases are associated with gene expression programs favoring SARS-CoV-2 entry and severity

L. T. Bui, N. I. Winters, M.-I. Chung, C. Joseph, A. J. Gutierrez, A. C. Habermann, T. S. Adams, J. C. Schupp, S. Poli, L. M. Peter, C. J. Taylor, J. B. Blackburn, B. W. Richmond, A. G. Nicholson, D. Rassl, , Human Cell Atlas Lung Biological Network

The paper analyzes single-cell transcriptomes from healthy and chronic lung disease lungs to identify gene expression programs linked to SARS-CoV-2 entry and severity.

bioRxiv · Oct 20, 2020Cell Biology

Gene expression and in situ protein profiling of candidate SARS-CoV-2 receptors in human airway epithelial cells and lung tissue

J. A. Aguiar, B. J.-M. Tremblay, M. J. Mansfield, O. Woody, B. Lobb, A. Banerjee, A. Chandiramohan, N. Tiessen, A. Dvorkin-Gheva, S. Revill, M. S. Miller, C. Carlsten, L. Organ, C. Joseph, A. John, , J. A. Hirota

The paper examines gene expression and protein localization of potential SARS-CoV-2 receptors in human airway epithelial cells and lung tissue, finding low ACE2 expression and confirming other candidate receptors.

bioRxiv · Apr 7, 2020Immunology