Author

A. Deka

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Immunology· 3 Bioengineering· 1

Signature Assays, Organisms & Methods

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Research Focus:differential expression· 3 Approach:experimental assay· 3 Research Focus:host-pathogen & immune response· 3 Organism:mouse· 3 Assay:bulk RNA-seq· 2 Research Focus:cell type annotation· 2 Organism:human· 2 Approach:statistical model· 2

Frequent Co-Authors

S. Basak· 3 shared M. Chawla· 2 shared V. Ahuja· 2 shared A. Awasthi· 1 shared A. K. Singh· 1 shared A. S. Ali· 1 shared B. Banoth· 1 shared B. Chatterjee· 1 shared

Activity by Year

2020-2024 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 26 mutual co-author links

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3 shared papersImmunology

S. Basak

Mutual co-authors in cluster

Author profile →

4 papers

Recent·Most cited

Modeling the impact of optimized airflow and sick pen management on the spread of infectious diseases in swine barns

M. Safari, C. Fleming, J. A. Galvis, A. Deka, F. Sanchez, G. Machado, C.-A. Yeh

This study models how optimized airflow and sick pen placement in swine barns affect disease spread, finding that ventilation adjustments and strategic pen placement can reduce disease transmission by up to 43.2%.

bioRxiv · Mar 13, 2024Bioengineering

Non-canonical NF-κB signaling promotes intestinal inflammation by restraining the tolerogenic β-catenin-Raldh2 axis in dendritic cells

A. Deka, N. Kumar, M. Chawla, N. Bhattacharya, A. S. Ali, S. Basu, Bhawna, U. Madan, S. Kumar, B. Das, D. Sengupta, A. Awasthi, S. Basak

bioRxiv · Dec 3, 2023 · PreprintImmunology

A hepatocyte-specific transcriptional program driven by Rela and Stat3 exacerbates experimental colitis in mice by modulating bile synthesis

Jyotsna, B. Sarkar, M. Yadav, A. Deka, M. Markandey, P. Sanyal, P. Nagarajan, N. Gaikward, V. Ahuja, D. Mohanty, S. Basak, R. S. Gokhale

bioRxiv · Sep 21, 2023Immunology

An epithelial Nfkb2 pathway exacerbates intestinal inflammation by supplementing latent RelA dimers to the canonical NF-κB module

M. Chawla, T. Mukherjee, A. Deka, B. Chatterjee, U. A. Sarkar, A. K. Singh, S. Kedia, B. Banoth, S. K. Biswas, V. Ahuja, S. Basak

The paper shows that noncanonical NF-κB signaling in intestinal epithelial cells enhances latent RelA dimers, amplifying canonical NF-κB activity and exacerbating intestinal inflammation in IBD.

bioRxiv · Nov 2, 2020Immunology