Author

A. Gope

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Microbiology· 2 Cancer Biology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Organism:human· 3 Organism:mouse· 3 Research Focus:host-pathogen & immune response· 2 Assay:proteomics· 2 Assay:bulk RNA-seq· 1 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell type annotation· 1

Frequent Co-Authors

A. Pal· 2 shared A. K. Mukhopadhyay· 1 shared B. C. Karmakar· 1 shared D. Ganguli· 1 shared D. Sarkar· 1 shared E. Mahapatra· 1 shared N. Nag· 1 shared P. Basak· 1 shared

Activity by Year

2021-2023 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 17 mutual co-author links

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2 shared papersCancer Biology

A. Pal

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

Metallo-protease Peptidase M84 from Bacillus altitudinis induces ROS dependent apoptosis in ovarian cancer cells by targeting PAR-1

N. Nag, T. Ray, R. Tapader, A. Gope, R. Das, E. Mahapatra, S. Saha, A. Pal, P. Prasad, S. Chatterjee, S. S. Roy, A. Pal

This paper identifies Peptidase M84 from Bacillus altitudinis as an apoptosis inducer in ovarian cancer cells via PAR-1 mediated oxidative stress, showing anti-tumor effects in vitro and in vivo.

bioRxiv · Sep 6, 2023 · PreprintCancer Biology

Glycyrrhizin, an inhibitor of HMGB1 induces autolysosomal degradation function and inhibits H. pylori infection

U. Khan, B. C. Karmakar, P. Basak, S. Paul, A. Gope, D. Sarkar, A. K. Mukhopadhyay, S. Dutta, S. Bhattacharya

This study shows glycyrrhizin, an HMGB1 inhibitor, restores lysosomal function and reduces H. pylori infection in gastric cells and mice.

bioRxiv · Aug 2, 2022 · PreprintMicrobiology

Macrophage cell lines and murine infection by Salmonella Typhi L-form bacteria

S. Das, D. Ganguli, S. Chakraborty, S. Chakraborty, A. Pal, A. Gope

This study shows that antibiotic treatment of Salmonella Typhi can generate L-form bacteria that infect macrophages and mice, suggesting a mechanism for antibiotic resistance and disease transmission.

bioRxiv · Dec 10, 2021 · PreprintMicrobiology