Author

A. Khosravi

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 1 Immunology· 1 Microbiology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Organism:human· 3 Research Focus:cell type annotation· 2 Assay:bulk RNA-seq· 1 Research Focus:differential expression· 1 Research Focus:host-pathogen & immune response· 1 Assay:imaging· 1 Assay:proteomics· 1

Frequent Co-Authors

A. Hargil· 1 shared A. Olin· 1 shared A. Yates· 1 shared A. Zareour· 1 shared A. Zarrabi· 1 shared C. K. Sen· 1 shared C. R. de Vries· 1 shared D. Liu· 1 shared

Activity by Year

2019-2024 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 17 mutual co-author links

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1 shared paperMicrobiology

A. Hargil

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

Assessment of Stiffness-Dependent Autophagosome Formation and Apoptosis in Embryonal Rhabdomyosarcoma Tumor Cells

S. Sezen, S. Adiguzel, A. Zareour, A. Khosravi, J. Gordon, S. Ghavami, A. Zarrabi

This study evaluates how matrix stiffness affects autophagosome formation and apoptosis in embryonal rhabdomyosarcoma cells using GelMA hydrogels.

bioRxiv · Mar 1, 2024 · PreprintCancer Biology

Filamentous Bacteriophage Delay Healing of Pseudomonas-Infected Wounds

M. S. Bach, C. R. de Vries, A. Khosravi, J. M. Sweere, M. Popescu, A. Hargil, J. D. Van Belleghem, G. Kaber, Q. Chen, E. B. Burgener, D. Liu, Q.-L. Tran, T. Dharmaraj, M. Birukova, V. Sunkari, , P. L. Bollyky

This study shows filamentous bacteriophage delays wound healing in Pseudomonas aeruginosa infections by inhibiting keratinocyte migration, supported by a patient cohort study.

bioRxiv · Mar 10, 2020Microbiology

Cell Generation Dynamics Underlying Naïve T Cell Homeostasis in Adult Humans

J. Mold, P. Reu, A. Olin, S. Bernard, J. Michaëlsson, S. Rane, A. Yates, A. Khosravi, M. Salehpour, G. Possnert, P. Brodin, J. Frisen

This study measures age-related changes in naive T cell turnover using 14C dating and identifies NF-κB signaling as critical for homeostatic proliferation.

bioRxiv · May 13, 2019 · PreprintImmunology