Author

A. Mathison

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 2

Signature Assays, Organisms & Methods

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Assay:bulk RNA-seq· 2 Research Focus:cell type annotation· 2 Organism:mouse· 2 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell signaling & trafficking· 1 Research Focus:differential expression· 1 Approach:experimental assay· 1 Research Focus:gene regulatory network inference· 1

Frequent Co-Authors

A. El kaoutari· 1 shared A. Salmonson· 1 shared A. Thomas· 1 shared C. Johnson· 1 shared C. L. Pin· 1 shared E. Jaune-Pons· 1 shared F. Mousavi· 1 shared G. A. Lomberk· 1 shared

Activity by Year

2023-2024 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 21 mutual co-author links

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1 shared paperCancer Biology

A. El kaoutari

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Author profile →

2 papers

Recent·Most cited

EHMT2 Inactivation in Pancreatic Epithelial Cells Shapes the Transcriptional Landscape and Inflammation Response of the Whole Pancreas

G. Pollin, A. Mathison, T. Milech de Assuncao, A. Thomas, L. Zeighami, A. Salmonson, H. Liu, G. Urrutia, P. Vankayala, S. J. Pandol, M. T. Zimmermann, J. L. Iovanna, V. X. Jin, R. A. Urrutia, G. A. Lomberk

bioRxiv · Mar 14, 2024Cancer Biology

EZH2 deletion does not impact acinar cell regeneration but restricts progression to pancreatic cancer in mice

E. Jaune-Pons, X. Wang, F. Mousavi, A. El kaoutari, K. Berger, C. Johnson, M. Martin, S. Aggarwal, S. Brar, K. Muhammad, P. Shooshtari, A. Mathison, N. Dusetti, R. Urrutia, G. Lomberk, C. L. Pin

This study shows EZH2 deletion in adult acinar cells does not block regeneration but limits PDAC progression by altering the tumor microenvironment and differentiation.

bioRxiv · Sep 25, 2023 · PreprintCancer Biology