A. P. Liu

This study shows that matrix confinement affects 3D spheroid sorting and collective migration, with sorting dependent on cell-cell adhesion and matrix resistance.

The paper develops an optical biosensor to measure membrane tension by repurposing an E. coli mechanosensitive channel with circularly permuted GFP.

This study shows that actin network architecture and lumenal viscosity regulate GUV deformability, with crosslinkers and dendritic cortex reducing deformability compared to actin filaments alone.

This study identifies compressive stress as a driver of unjamming transitions in breast cancer cell migration by showing that cell-cell adhesion, not epithelial-mesenchymal transition, mediates these changes.

The paper shows that actomyosin networks in lipid vesicles form ring and aster patterns, driving membrane constriction and deformation through myosin-driven remodeling.

This study develops a 2D platform to enhance contractile development and reproducibility in stem cell-derived cardiac muscle by controlling biomechanics and myofibrillar alignment.

This paper develops a cell-free system to study LINC complex assembly by reconstituting membrane proteins in lipid bilayers and shows its utility in investigating chemical and molecular factors affecting protein interactions.

This study shows myofibrillar structure variability in iPSC-derived cardiomyocytes affects contractile function and should be considered in disease modeling and drug testing.

The paper shows that actin crosslinker competition and sorting in confined environments lead to distinct actin network architectures in GUVs.

The paper identifies a third mechanism of intracellular GPCR signaling independent of ligands and endocytosis, mediated by β-arrestin-1.

The paper shows epsin's structured and unstructured domains work together to support clathrin-mediated endocytosis under high membrane tension.

The paper shows that acute plasma membrane tension decrease triggers macropinocytosis through PLD2 activation and phosphatidic acid production in myoblasts.

This study shows PTEN regulates clathrin-coated pit dynamics by modulating PI(3,4,5)P3 levels, promoting short-lived signaling-capable CCPs in cancer cells.