Author

A. Rosa Campos

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

Click to filter author papers

Cell Biology· 2 Cancer Biology· 1

Signature Assays, Organisms & Methods

Click to filter · or open tag

Approach:experimental assay· 3 Organism:mouse· 3 Assay:proteomics· 3 Assay:bulk RNA-seq· 1 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell signaling & trafficking· 1 Research Focus:cell type annotation· 1 Research Focus:cell-cell communication· 1

Frequent Co-Authors

A. Bagchi· 1 shared A. C. McQuattie-Pimentel· 1 shared A. Deshpande· 1 shared A. V. Misharin· 1 shared B. Chen· 1 shared C. J. Eaves· 1 shared C.-C. Yang· 1 shared D. A. Wolf· 1 shared

Activity by Year

2019-2020 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 14 mutual co-author links

Click any collaborator node to inspect or filter shared papers

Focal author (center)Selected co-authorORCID co-authorUnlinked name

Curved chords = mutual co-authorship

1 shared paperCancer Biology

A. Bagchi

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

Cancer BiologybioRxivAug 31, 2020

A JAK/STAT-Mediated Inflammatory Signaling Cascade Drives Oncogenesis In AF10-Rearranged AML

B. Chen, A. Deshpande, K. O. Barbosa…·A. Deshpande

This study identifies JAK1 as a therapeutic target in AF10-rearranged AML by showing its role in JAK/STAT-mediated inflammatory signaling and oncogenesis.

DOI 10.1182/blood.2020009023

mousebulk RNA-seq

𝕏 PostBluesky

Cell BiologybioRxivPreprintSep 14, 2019

Proteostasis and Energetics as Proteome Hallmarks of Aging and Influenza Challenge in Pulmonary Disease

S. Loguercio, D. M. Hutt, A. Rosa Campos…·W. E. Balch

This study examines proteome changes in alveolar macrophages and type 2 cells from young and old mice before and after influenza infection, revealing age-related proteostasis and mitochondrial dysfunction that worsen with infection.

DOI 10.1101/769737

mouseproteomics

𝕏 PostBluesky

Cell BiologybioRxivJul 5, 2019

Crosstalk between eIF2α and eEF2 phosphorylation pathways optimizes translational arrest in response to oxidative stress

M. Sanchez, Y. Lin, C.-C. Yang…·D. A. Wolf

This study shows that oxidative stress causes translational arrest through eEF2 phosphorylation, not eIF2, and reveals crosstalk between eIF2 and eEF2 pathways in mouse cells.

DOI 10.1016/j.isci.2019.09.031

mouseproteomics

𝕏 PostBluesky