Author

A. Sayed

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cell Biology· 2 Bioengineering· 1

Signature Assays, Organisms & Methods

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Research Focus:cell type annotation· 3 Approach:experimental assay· 2 Organism:mouse· 2 Assay:bulk RNA-seq· 1 Research Focus:cell signaling & trafficking· 1 Research Focus:cell-cell communication· 1 Research Focus:clustering· 1 Research Focus:development & morphogenesis· 1

Frequent Co-Authors

G. Marazzi· 2 shared J.-S. Hulot· 2 shared M. Valente· 2 shared B. Hoareau· 1 shared D. D. A. Sassoon· 1 shared D. Li· 1 shared D. Ollitrault· 1 shared D. Sassoon· 1 shared

Activity by Year

2021-2021 · peak 3/yr

Co-Author Collaboration Network

10 top collaborators · 23 mutual co-author links

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2 shared papersCell Biology

G. Marazzi

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

Hypoxia promotes a perinatal-like progenitor state in the adult murine epicardium

A. Sayed, S. Turoczi, F. Soares-da-Silva, G. Marazzi, J.-S. Hulot, D. D. A. Sassoon, M. Valente

This study shows that hypoxia reactivates progenitor-like properties in adult murine epicardial cells, promoting endothelial fate and multilineage differentiation potential.

bioRxiv · Sep 16, 2021Cell Biology

Fibroadipogenic Progenitors contribute to microvascular repair during skeletal muscle regeneration

D. Ollitrault, V. Buffa, R. Correra, A. Sayed, B. Hoareau, S. Pohle-Kronawitter, S. Stricker, J.-S. Hulot, M. Valente, G. Marazzi, D. Sassoon

This study shows that fibroadipogenic progenitors (FAPs) differentiate into endothelial cells during skeletal muscle regeneration in response to hypoxia and glucose deprivation.

bioRxiv · Sep 6, 2021 · PreprintCell Biology

Amelioration of hemophilia B through CRISPR/Cas9 induced homology-independent targeted integration

X. Chen, X. Niu, Y. Liu, R. Zheng, L. Wang, L. Yang, J. Lu, S. Yin, Y. Shao, Y. Wei, J. Pan, A. Sayed, X. Ma, M. Liu, F. Jing, , D. Li

This study uses CRISPR/Cas9 to induce homology-independent targeted integration in a rat hemophilia B model, achieving therapeutic correction with 3.66% knock-in efficiency and normalization of clotting time.

bioRxiv · Mar 18, 2021 · PreprintBioengineering