Author

Andreas Unger

University of MünsterPrimary ORCID profile (0000-0002-6538-8726 · 1 paper)Unclaimed ORCID profile · follow to get notified of new preprints

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Same name, identified by ORCID

Andreas UngeriD1 paper · Technical University of Denmark

Biology Disciplines

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Physiology· 2 Cell Biology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Organism:mouse· 3 Research Focus:cell type annotation· 2 Research Focus:differential expression· 2 Research Focus:gene regulatory network inference· 2 Organism:human· 2 Assay:ATAC-seq· 1 Assay:bulk RNA-seq· 1

Frequent Co-Authors

Wolfgang Albrecht LinkeiD· 2 shared Ahmed WagdiiD· 1 shared Alice MarinoiD· 1 shared Anne HelfeniD· 1 shared Annika J. Klotz· 1 shared B. Bearzatto· 1 shared Branimir Berečić· 1 shared Caroline BouziniD· 1 shared

Activity by Year

2025-2025 · peak 3/yr

Co-Author Collaboration Network

10 top collaborators · 15 mutual co-author links

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2 shared papersPhysiology

Wolfgang Albrecht LinkeORCID

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Author profile →

3 papers

Recent·Most cited

skNAC is a Key Driver of Cardiomyocyte Integrity Against Pathological Cardiac Hypertrophy and Heart Failure

Laura Guilbert, Justine Dontaine, Natacha Fourny, Ettore Vanni, Michele Russo, Hugo Vanderroost, Jade Dron, Jérôme Ambroise, Hrag Esfahani, Caroline Bouzin, Younès Achouri, Emilie Hendrickx, Nassiba Menghoum, B. Bearzatto, D. Vertommen, , Luc Bertrand

This study identifies skNAC as critical for maintaining cardiomyocyte integrity and preventing heart failure by regulating proteome homeostasis and metabolic adaptation during pathological hypertrophy.

bioRxiv · Oct 23, 2025 · PreprintPhysiology

Peroxisome protein import deficiency causes heart failure in mouse and human

Julia Hofhuis, Malte Tiburcy, Yelena Hartmann, Kristina Bersch, Ahmed Wagdi, Óscar Gutiérrez-Gutiérrez, Richard Solano, Leonie Thiele, Maya Walper, Mona Göbel, Wolfgang Hübner, Branimir Berečić, Marilu Casini, Petros Tirilomis, Andreas Unger, , Sven Thoms

This study shows that peroxisome dysfunction in mice and engineered human myocardium leads to heart failure through metabolic impairment and disrupted cellular respiration.

bioRxiv · Oct 13, 2025 · PreprintCell Biology

Titin cleavage impairs cardiac mechanical connectivity to drive diastolic failure and fibrosis

Johanna K. Freundt, Paulina Hartmann, Christine M. Loescher, Andreas Unger, Franziska Koser, Annika J. Klotz, Lydia Wachsmuth, Susanne S. Hille, Michaela M. Door, Anne Helfen, Richard Holtmeier, Cornelius Faber, Jonathan A. Kirk, Verena Hoerr, Oliver J Mueller, Wolfgang Albrecht Linke

This study shows that titin cleavage in mice causes concentric cardiac remodeling, diastolic dysfunction, and fibrosis by disrupting mechanical connectivity.

bioRxiv · Apr 21, 2025 · PreprintPhysiology