Author

Anuradha R. Iyer

Cardiovascular Research Center

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cell Biology· 1 Physiology· 1

Signature Assays, Organisms & Methods

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Research Focus:cell signaling & trafficking· 2 Organism:mouse· 2 Research Focus:cell type annotation· 1 Research Focus:development & morphogenesis· 1 Approach:experimental assay· 1 Approach:genetic perturbation / CRISPR· 1 Research Focus:host-pathogen & immune response· 1 Assay:proteomics· 1

Frequent Co-Authors

Hannah E. VorndraniD· 2 shared Jeffrey L. BrodskyiD· 2 shared Teresa M. BuckiD· 2 shared Aidan W. PorteriD· 1 shared Allison E. Marciszyn· 1 shared Ashley R. Flory· 1 shared Chris Melendez-Suchi· 1 shared Christopher J. GuerrieroiD· 1 shared

Activity by Year

2024-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 33 mutual co-author links

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2 shared papersPhysiology

Hannah E. VorndranORCID

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2 papers

Recent·Most cited

PhysiologyAmerican Journal of Physiology-Renal PhysiologyPeer-reviewedOct 21, 2026

An endoplasmic reticulum resident molecular chaperone, GRP170, prevents stress-induced glomerular injury

Aidan W. Porter, Allison E. Marciszyn, Hannah E. Vorndran…·Teresa M. Buck

This study shows that GRP170 protects podocytes from stress-induced glomerular injury by modulating the UPR, as demonstrated in a podocyte-specific knockout mouse model.

DOI 10.1152/ajprenal.90091.2026

mouseexperimental assay

𝕏 PostBluesky

Cell BiologyMolecular Biology of the CellPeer-reviewedMar 6, 2024

Loss of Grp170 results in catastrophic disruption of endoplasmic reticulum functions

Melissa J. Mann, Chris Melendez-Suchi, Hannah E. Vorndran…·Teresa M. Buck

This study shows that loss of Grp170 causes ER dysfunction, including BiP misregulation and UPR activation, leading to cell death in mouse embryonic fibroblasts.

13 citations

mouseproteomics

𝕏 PostBluesky