Author

B. E. Windle

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 2

Signature Assays, Organisms & Methods

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Research Focus:cancer & tumor microenvironment· 2 Approach:experimental assay· 2 Organism:human· 2 Assay:bulk RNA-seq· 1 Assay:proteomics· 1

Frequent Co-Authors

B. Ding· 1 shared B. Hu· 1 shared D. Basu· 1 shared D. P. Kelly· 1 shared E. A. White· 1 shared E. Oduah· 1 shared H. Harada· 1 shared H. Nakagawa· 1 shared

Activity by Year

2022-2024 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 21 mutual co-author links

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1 shared paperCancer Biology

B. Ding

Mutual co-authors in cluster

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2 papers

Recent·Most cited

Oncogenic Mutant p53 Sensitizes Non-Small Cell Lung Cancer Cells to Proteasome Inhibition via Oxidative Stress-Dependent Induction of Mitochondrial Apoptosis

K. K. Chougoni, V. Neely, B. Ding, E. Oduah, V. Lam, B. Hu, J. E. Koblinski, B. E. Windle, S. P. Deb, S. Deb, S. K. Radhakrishnan, H. Harada, S. R. Grossman

This study shows proteasome inhibitors are more effective against NSCLC cells with mutant p53 by inducing oxidative stress and mitochondrial apoptosis, suggesting a new therapeutic strategy.

bioRxiv · Feb 22, 2024Cancer Biology

Human papilloma virus E6 regulates therapy responses in oropharyngeal cancer by repressing the PGC-1α/ERRα axis

M. K. Sannigrahi, P. Rajagopalan, L. Lai, X. Liu, V. Sahu, H. Nakagawa, J. B. Jalaly, R. M. Brody, I. M. Morgan, B. E. Windle, X. Wang, P. A. Gimotty, D. P. Kelly, E. A. White, D. Basu

This study shows that HPV E6 reduces mitochondrial antioxidant capacity and therapy resistance in oropharyngeal cancer by suppressing the PGC-1α/ERRα pathway.

bioRxiv · May 4, 2022 · PreprintCancer Biology