Author

B. Kiens

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

Click to filter author papers

Physiology· 3 Cancer Biology· 1

Signature Assays, Organisms & Methods

Click to filter · or open tag

Approach:experimental assay· 4 Research Focus:metabolism & bioenergetics· 4 Organism:mouse· 4 Assay:metabolomics· 3 Research Focus:gene regulatory network inference· 2 Organism:human· 2 Assay:proteomics· 2 Research Focus:cell signaling & trafficking· 1

Frequent Co-Authors

A. M. Fritzen· 3 shared L. L. V. Moller· 2 shared L. Sylow· 2 shared M. Kleinert· 2 shared A. Artati· 1 shared A. B. Klein· 1 shared A. Cebrian-Serrano· 1 shared A. Gudiksen· 1 shared

Activity by Year

2019-2023 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 24 mutual co-author links

Click any collaborator node to inspect or filter shared papers

Focal author (center)Selected co-authorORCID co-authorUnlinked name

Curved chords = mutual co-authorship

3 shared papersPhysiology

A. M. Fritzen

Mutual co-authors in cluster

Author profile →

4 papers

Recent·Most cited

Pantothenate Kinase 4 controls efficient skeletal muscle energy substrate metabolism via acetyl-CoA

A. Miranda-Cervantes, A. M. Fritzen, S. H. Raun, O. Hodek, L. L. V. Moller, K. Johann, L. Deisen, P. Gregorevic, A. Gudiksen, A. Artati, J. Adamski, N. R. Andersen, P. Schjerling, A. Cebrian-Serrano, M. Jaehnert, , M. Kleinert

This study identifies PanK4 as a regulator of skeletal muscle energy metabolism, showing its role in acetyl-CoA regulation and metabolic flexibility.

bioRxiv · Aug 14, 2023 · PreprintPhysiology

Pharmacological but not physiological GDF15 suppresses feeding and the motivation to exercise

A. B. Klein, T. S. Nicolaisen, N. Ortenblad, K. D. Gejl, R. Jensen, A. M. Fritzen, E. L. Larsen, K. Karstoft, H. E. Poulsen, T. Morville, R. E. Sahl, J. W. Helge, J. Lund, S. Falk, M. Lyngbaek, , C. Clemmensen

The paper shows that exercise increases circulating GDF15 levels, but this physiological increase does not suppress appetite or exercise motivation like pharmacological GDF15.

bioRxiv · Oct 23, 2020Physiology

Cancer causes metabolic perturbations associated with reduced insulin-stimulated glucose uptake in peripheral tissues and impaired muscle microvascular perfusion

X. Han, S. Raun, M. Carlsson, K. A. Sjoberg, C. Henriquez-Olguin, M. Ali, A. Lundsgaard, A. M. Fritzen, L. L. V. Moller, Z. Li, J. Li, T. E. Jensen, B. Kiens, L. Sylow

This study shows cancer reduces insulin-stimulated glucose uptake in muscle and adipose tissue and impairs muscle microvascular perfusion in mice.

bioRxiv · Dec 28, 2019Cancer Biology

Restriction of essential amino acids dictates the systemic response to dietary protein dilution

Y. W. Yap, P. M. Rusu, A. Y. Chan, B. C. Fam, A. Jungmann, S. M. Solon-Biet, C. K. Barlow, D. J. Creek, C. Huang, R. B. Schittenhelm, B. Morgan, D. Schmoll, B. Kiens, M. D. Piper, M. Heikenwalder, , A. J. Rose

This study shows that restricting essential amino acids, not non-essential ones, drives systemic metabolic responses to dietary protein dilution and genetic amino acid transport loss.

bioRxiv · Nov 20, 2019 · PreprintPhysiology