Author

B. Uyar

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cell Biology· 2 Biochemistry· 1 Cancer Biology· 1

Signature Assays, Organisms & Methods

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Organism:human· 3 Assay:bulk RNA-seq· 2 Research Focus:cell signaling & trafficking· 2 Research Focus:cell type annotation· 2 Research Focus:chromatin & epigenetic regulation· 2 Research Focus:development & morphogenesis· 2 Approach:experimental assay· 2 Organism:C. elegans· 1

Frequent Co-Authors

A. Akalin· 4 shared A. B. Tufan· 1 shared A. Baranovskii· 1 shared A. Krause· 1 shared B. Tursun· 1 shared B. Vitrinel· 1 shared C. A. Schmitt· 1 shared C. Scheidereit· 1 shared

Activity by Year

2017-2021 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 21 mutual co-author links

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4 shared papersCell Biology

A. Akalin

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Author profile →

4 papers

Recent·Most cited

Identifying tumor cells at the single cell level

J. Dohmen, A. Baranovskii, B. Uyar, J. Ronen, V. Franke, A. Akalin

This paper introduces Ikarus, a machine learning pipeline that identifies tumor cells in single-cell data with high sensitivity and specificity across multiple datasets.

bioRxiv · Oct 15, 2021Cancer Biology

A novel IKK- and proteasome-independent mechanism of RelA activation triggers senescence associated secretome via transcriptional repression of NFKBIA

M. Kolesnichenko, N. Mikuda, U. E. Höpken, M. Milanovic, A. B. Tufan, B. Uyar, W. Sun, K. Schleich, L. von Hoff, M. Willenbrock, I. Krahn, S. Jungmann, M. Hinz, A. Akalin, S. Lee, , C. Scheidereit

The paper identifies a novel IKK- and proteasome-independent mechanism of RelA activation in senescent cells that drives the senescence-associated secretome through transcriptional repression of NFKBIA.

bioRxiv · Dec 19, 2019Cell Biology

The conserved histone chaperone LIN-53 links lifespan and healthspan regulation in C. elegans

S. Muethel, B. Uyar, M. He, A. Krause, B. Vitrinel, S. Bulut, D. Vasiljevic, A. Akalin, S. Kempa, B. Tursun

This study shows LIN-53 links lifespan and healthspan in C. elegans by regulating chromatin and trehalose levels, with implications for evolutionary conservation.

bioRxiv · Feb 2, 2019Cell Biology

Mutations In Disordered Regions Cause Disease By Creating Endocytosis Motifs

K. Meyer, B. Uyar, M. Kirchner, J. Cheng, A. Akalin, M. Selbach

The paper identifies mutations in disordered regions that create dileucine motifs, leading to clathrin-dependent trafficking and disease.

bioRxiv · May 24, 2017Biochemistry