Author

C. C. Fronick

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

Click to filter author papers

Cancer Biology· 3

Signature Assays, Organisms & Methods

Click to filter · or open tag

Assay:bulk RNA-seq· 3 Research Focus:cell type annotation· 3 Research Focus:differential expression· 3 Organism:human· 3 Research Focus:cell-cell communication· 2 Assay:scRNA-seq· 2 Assay:spatial transcriptomics· 2 Approach:statistical model· 2

Frequent Co-Authors

R. S. Fulton· 3 shared C. Oh· 2 shared C.-K. Mo· 2 shared E. L. Appelbaum· 2 shared E. Storrs· 2 shared H. Sun· 2 shared H. Zhu· 2 shared J. M. Herndon· 2 shared

Activity by Year

2021-2023 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 45 mutual co-author links

Click any collaborator node to inspect or filter shared papers

Focal author (center)Selected co-authorORCID co-authorUnlinked name

Curved chords = mutual co-authorship

3 shared papersCancer Biology

R. S. Fulton

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

Differential chromatin accessibility and transcriptional dynamics define breast cancer subtypes and their lineages

M. D. Iglesia, R. G. Jayasinghe, S. Chen, N. V. Terekhanova, J. M. Herndon, E. Storrs, A. Karpova, D. C. Zhou, N. Naser Al Deen, A. T. Shinkle, R. J.-H. Lu, W. Caravan, A. Houston, Y. Zhao, K. Sato, , Marge

This study identifies breast cancer subtypes and their lineage origins by analyzing chromatin accessibility and transcriptional dynamics using multi-omic techniques.

bioRxiv · Oct 31, 2023Cancer Biology

Immunosuppression and Outcomes in Acute Myeloid Leukemia

F. Ferraro, C. Miller, K. Christensen, N. M. Helton, M. O'Laughlin, C. C. Fronick, R. S. Fulton, J. Kohlschmidt, A.-K. Eisfeld, C. D. Bloomfield, S. M. Ramakrishnan, R. B. Day, L. D. Wartman, G. L. Uy, J. S. Welch, , T. J. Ley

This study identifies immunosuppressive features in AML cells associated with poor outcomes in patients treated with standard chemotherapy.

bioRxiv · Sep 3, 2021 · PreprintCancer Biology

Spatial drivers and pre-cancer populations collaborate with the microenvironment in untreated and chemo-resistant pancreatic cancer

D. Cui Zhou, R. G. Jayasinghe, J. M. Herndon, E. Storrs, C.-K. Mo, Y. Wu, R. S. Fulton, M. A. Wyczalkowski, C. C. Fronick, L. A. Fulton, L. Thammavong, K. Sato, H. Zhu, H. Sun, L.-B. Wang, , Ch

This paper uses spatial single-cell omics to analyze PDAC tumors and microenvironment, identifying key populations and interactions that may inform treatment strategies.

bioRxiv · Jan 13, 2021Cancer Biology