Author

C. U. Awah

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 3

Signature Assays, Organisms & Methods

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Organism:human· 3 Research Focus:drug response / drug discovery· 2 Approach:experimental assay· 2 Assay:bulk RNA-seq· 1 Research Focus:cancer & tumor microenvironment· 1 Assay:CRISPR screen· 1 Approach:genetic perturbation / CRISPR· 1 Research Focus:transcription & RNA processing· 1

Frequent Co-Authors

J. Winter· 3 shared O. Ogunwobi· 2 shared A. Ahmed· 1 shared A. M. Sonabend· 1 shared A. Mahajan· 1 shared B. Gyorffy· 1 shared C. M. Mazdoom· 1 shared C. Park· 1 shared

Activity by Year

2019-2021 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 31 mutual co-author links

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3 shared papersCancer Biology

J. Winter

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Author profile →

3 papers

Recent·Most cited

Cancer BiologybioRxivAug 11, 2021

Genome scale CRISPR Cas9a knockout screen reveals genes that controls glioblastoma susceptibility to the alkylating agent temozolomide

C. U. Awah, J. Winter, O. Ogunwobi

This study uses a genome-wide CRISPR-Cas9a knockout screen to identify genes influencing glioblastoma resistance to temozolomide.

DOI 10.1080/26895293.2021.2024895

humanCRISPR screen

𝕏 PostBluesky

Cancer BiologybioRxivAug 11, 2021

NSUN6, an RNA methyltransferase of 5-mC controls glioblastoma response to Temozolomide (TMZ) via NELFB and RPS6KB2 interaction.

C. U. Awah, J. Winter, C. M. Mazdoom, O. Ogunwobi

This study identifies NSUN6 as an RNA methyltransferase that regulates glioblastoma response to temozolomide through 5mC-mediated control of NELFB and RPS6KB2.

DOI 10.1080/15384047.2021.1990631

humanbulk RNA-seq

𝕏 PostBluesky

Cancer BiologybioRxivPreprintApr 25, 2019

Interaction between DNA damage response, translation and apoptosome determines cancer susceptibility to TOP2 poisons

C. U. Awah, L. Chen, M. Bansal…·A. M. Sonabend

The paper identifies genes involved in DNA damage response and translation that influence susceptibility to TOP2 poisons in gliomas and other cancers.

DOI 10.1101/614024

humanwhole-genome sequencing

𝕏 PostBluesky