Author

D. Blakemore

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 1 Cell Biology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Assay:bulk RNA-seq· 1 Research Focus:cell type annotation· 1 Research Focus:chromatin & epigenetic regulation· 1 Research Focus:differential expression· 1 Organism:human· 1 Organism:mouse· 1 Approach:statistical model· 1

Frequent Co-Authors

C. Ward· 2 shared P. Garcia· 2 shared A. Gambus· 1 shared D. S. Walton· 1 shared E. Gonzalez· 1 shared E. Petermann· 1 shared F. Grebien· 1 shared G. Murphy· 1 shared

Activity by Year

2020-2020 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 30 mutual co-author links

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2 shared papersCell Biology

C. Ward

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Author profile →

2 papers

Recent·Most cited

MYBL2 regulates ATM to control replication initiation and prevent replication stress in pluripotent stem cells

D. Blakemore, R. Almaghrabi, N. Vilaplana, E. Gonzalez, M. Moya, C. Ward, G. Murphy, A. Gambus, E. Petermann, G. Stewart, P. Garcia

This study shows MYBL2 activates ATM to suppress replication stress in ESCs, with loss of MYBL2 or ATM causing replication fork slowing and increased origin firing.

bioRxiv · Jun 4, 2020Cell Biology

Ablation of MYB-dependent leukaemia phenotype in MLL-driven AML correlates with increased expression of MAFB.

C. Ward, P. Cauchy, D. S. Walton, M. L. Clarke, D. Blakemore, F. Grebien, P. Garcia, J. Frampton, G. Volpe

The paper shows that MYB ablation in MLL-driven AML increases MAFB expression, while this effect is absent in AML with complex karyotypes.

bioRxiv · May 27, 2020 · PreprintCancer Biology