Author

E. Petermann

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cell Biology· 2 Cancer Biology· 1

Signature Assays, Organisms & Methods

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Research Focus:chromatin & epigenetic regulation· 3 Approach:experimental assay· 3 Research Focus:cell type annotation· 1 Assay:DNA methylation· 1 Organism:human· 1 Organism:mouse· 1

Frequent Co-Authors

G. Ronson· 2 shared G. Stewart· 2 shared A. Aitsiankova· 1 shared A. Gambus· 1 shared A. J. Garvin· 1 shared A. K. Walker· 1 shared A. L. Piberger· 1 shared A. Olsen· 1 shared

Activity by Year

2018-2024 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 18 mutual co-author links

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2 shared papersCancer Biology

G. Ronson

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

USP50 suppresses alternative RecQ helicase use and deleterious DNA2 activity during replication.

H. L. Mackay, H. R. Stone, K. Ellis, G. Ronson, K. Starowicz, A. J. Garvin, P. Van Eijk, A. Aitsiankova, S. Vijayendran, J. Beesley, E. Petermann, E. Brown, R. M. Densham, S. H. Reed, F. Dobbs, , J. R. Morris

This study identifies USP50 as a ubiquitin-binding protein that regulates helicase and nuclease activities during DNA replication to prevent fork collapse and maintain genomic stability.

bioRxiv · Jan 10, 2024Cancer Biology

MYBL2 regulates ATM to control replication initiation and prevent replication stress in pluripotent stem cells

D. Blakemore, R. Almaghrabi, N. Vilaplana, E. Gonzalez, M. Moya, C. Ward, G. Murphy, A. Gambus, E. Petermann, G. Stewart, P. Garcia

This study shows MYBL2 activates ATM to suppress replication stress in ESCs, with loss of MYBL2 or ATM causing replication fork slowing and increased origin firing.

bioRxiv · Jun 4, 2020Cell Biology

PARP1 and PARP2 stabilise replication forks at base excision repair intermediates through Fbh1-dependent Rad51 regulation

G. Ronson, A. L. Piberger, M. Higgs, A. Olsen, G. Stewart, P. McHugh, E. Petermann, N. Lakin

This study shows PARP1 and PARP2 stabilize replication forks during BER through Fbh1-dependent Rad51 regulation, revealing their distinct and linked roles in DNA repair.

bioRxiv · Jan 4, 2018Cell Biology