Author

D. Collier

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Microbiology· 3

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Research Focus:host-pathogen & immune response· 3 Organism:human· 3 Assay:bulk RNA-seq· 1 Assay:whole-genome sequencing· 1

Frequent Co-Authors

B. Meng· 3 shared G. Papa· 3 shared I. Ferreira· 3 shared A. Abdullahi· 2 shared J. Hyeon Lee· 2 shared L. James· 2 shared O. Charles· 2 shared P. Mlcochova· 2 shared

Activity by Year

2020-2021 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 45 mutual co-author links

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3 shared papersMicrobiology

B. Meng

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

SARS-CoV-2 Omicron spike mediated immune escape, infectivity and cell-cell fusion

B. Meng, I. Ferreira, A. Abdullahi, N. Goonawardane, A. Saito, I. Kimura, D. Yamasoba, S. A. Kemp, G. Papa, S. Fatihi, S. Rathore, P. Perera Gerba, T. Ikeda, M. Toyoda, T. S. Tan, , S. Zepeda

This study investigates the immune escape, infectivity, and cell-cell fusion properties of SARS-CoV-2 Omicron BA.1 compared to Delta, showing higher ACE2 affinity and antibody evasion but similar antiviral drug efficacy.

bioRxiv · Dec 17, 2021Microbiology

SARS-CoV-2 B.1.617.2 Delta variant replication, sensitivity to neutralising antibodies and vaccine breakthrough

P. Mlcochova, S. A. Kemp, M. Shanker Dhar, G. Papa, B. Meng, S. Mishra, C. Whittaker, T. Mellan, I. Ferreira, R. Datir, D. Collier, S. Singh, R. Pandey, K. Ponnusamy, V. S. Radhakrishnan, , Saliba

This study evaluates the replication, antibody sensitivity, and vaccine efficacy of the SARS-CoV-2 B.1.617.2 Delta variant compared to other variants and vaccine-elicited immunity.

bioRxiv · May 8, 2021 · PreprintMicrobiology

Recurrent emergence and transmission of a SARS-CoV-2 Spike deletion H69/V70

S. Kemp, B. Meng, I. Ferreira, R. Datir, W. Harvey, D. Collier, COVID-19 Genomics UK (COG-UK) consortium, G. Papa, A. Carabelii, S. Lytras, J. Kenyon, A. Lever, L. James, D. L. Robertson, R. K. Gupta

This paper identifies a recurrent SARS-CoV-2 Spike deletion (H69/V70) that increases infectivity and compensates for RBD mutations, suggesting it may facilitate immune escape and variant emergence.

bioRxiv · Dec 14, 2020Microbiology